| pubmed-article:17185353 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:17185353 | lifeskim:mentions | umls-concept:C0035647 | lld:lifeskim |
| pubmed-article:17185353 | lifeskim:mentions | umls-concept:C0006819 | lld:lifeskim |
| pubmed-article:17185353 | lifeskim:mentions | umls-concept:C0028351 | lld:lifeskim |
| pubmed-article:17185353 | lifeskim:mentions | umls-concept:C0442805 | lld:lifeskim |
| pubmed-article:17185353 | lifeskim:mentions | umls-concept:C0450254 | lld:lifeskim |
| pubmed-article:17185353 | pubmed:issue | 8 | lld:pubmed |
| pubmed-article:17185353 | pubmed:dateCreated | 2007-7-12 | lld:pubmed |
| pubmed-article:17185353 | pubmed:abstractText | Campylobacter jejuni can cause a spectrum of diseases in humans, ranging from enteritis and diarrhoea to severe inflammation, profuse bloody diarrhoea and chronic relapsing infection. Norepinephrine (NE) levels in the intestine increase under conditions of stress and trauma, and are thought to result in spill over of NE into the intestinal lumen. NE is known to stimulate the growth of a range of bacterial species, and to increase the pathogenicity of Escherichia coli. | lld:pubmed |
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| pubmed-article:17185353 | pubmed:language | eng | lld:pubmed |
| pubmed-article:17185353 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:17185353 | pubmed:citationSubset | AIM | lld:pubmed |
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| pubmed-article:17185353 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:17185353 | pubmed:month | Aug | lld:pubmed |
| pubmed-article:17185353 | pubmed:issn | 0017-5749 | lld:pubmed |
| pubmed-article:17185353 | pubmed:author | pubmed-author:WilliamsP HPH | lld:pubmed |
| pubmed-article:17185353 | pubmed:author | pubmed-author:HumphreyT JTJ | lld:pubmed |
| pubmed-article:17185353 | pubmed:author | pubmed-author:TaylorA HAH | lld:pubmed |
| pubmed-article:17185353 | pubmed:author | pubmed-author:JepsonM AMA | lld:pubmed |
| pubmed-article:17185353 | pubmed:author | pubmed-author:KetleyJJ | lld:pubmed |
| pubmed-article:17185353 | pubmed:author | pubmed-author:CoganT ATA | lld:pubmed |
| pubmed-article:17185353 | pubmed:author | pubmed-author:HIRTEW EWE | lld:pubmed |
| pubmed-article:17185353 | pubmed:author | pubmed-author:ThomasA OAO | lld:pubmed |
| pubmed-article:17185353 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:17185353 | pubmed:volume | 56 | lld:pubmed |
| pubmed-article:17185353 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:17185353 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:17185353 | pubmed:pagination | 1060-5 | lld:pubmed |
| pubmed-article:17185353 | pubmed:dateRevised | 2010-9-14 | lld:pubmed |
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| pubmed-article:17185353 | pubmed:meshHeading | pubmed-meshheading:17185353... | lld:pubmed |
| pubmed-article:17185353 | pubmed:year | 2007 | lld:pubmed |
| pubmed-article:17185353 | pubmed:articleTitle | Norepinephrine increases the pathogenic potential of Campylobacter jejuni. | lld:pubmed |
| pubmed-article:17185353 | pubmed:affiliation | Division of Veterinary Pathology, Infection and Immunity, School of Clinical Veterinary Science, University of Bristol, Langford, Bristol BS40 5DU, UK. tristan.cogan@bristol.ac.uk | lld:pubmed |
| pubmed-article:17185353 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:17185353 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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