| pubmed-article:17138581 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:17138581 | lifeskim:mentions | umls-concept:C0206524 | lld:lifeskim |
| pubmed-article:17138581 | lifeskim:mentions | umls-concept:C1293097 | lld:lifeskim |
| pubmed-article:17138581 | lifeskim:mentions | umls-concept:C0015219 | lld:lifeskim |
| pubmed-article:17138581 | lifeskim:mentions | umls-concept:C0441712 | lld:lifeskim |
| pubmed-article:17138581 | pubmed:issue | 4 | lld:pubmed |
| pubmed-article:17138581 | pubmed:dateCreated | 2007-3-26 | lld:pubmed |
| pubmed-article:17138581 | pubmed:abstractText | The continuity of chloroplasts is maintained by division of pre-existing chloroplasts. Chloroplasts originated as bacterial endosymbionts; however, the majority of bacterial division factors are absent from chloroplasts and the eukaryotic host has added several new components. For example, the ftsZ gene has been duplicated and modified, and the Min system has retained MinE and MinD but lost MinC, acquiring at least one new component ARC3. Further, the mechanism has evolved to include two members of the dynamin protein family, ARC5 and FZL, and plastid-dividing (PD) rings were most probably added by the eukaryotic host. | lld:pubmed |
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