| pubmed-article:17118179 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:17118179 | lifeskim:mentions | umls-concept:C0015024 | lld:lifeskim |
| pubmed-article:17118179 | lifeskim:mentions | umls-concept:C0017337 | lld:lifeskim |
| pubmed-article:17118179 | lifeskim:mentions | umls-concept:C0026882 | lld:lifeskim |
| pubmed-article:17118179 | lifeskim:mentions | umls-concept:C0010744 | lld:lifeskim |
| pubmed-article:17118179 | lifeskim:mentions | umls-concept:C1521991 | lld:lifeskim |
| pubmed-article:17118179 | lifeskim:mentions | umls-concept:C0032150 | lld:lifeskim |
| pubmed-article:17118179 | lifeskim:mentions | umls-concept:C0733511 | lld:lifeskim |
| pubmed-article:17118179 | lifeskim:mentions | umls-concept:C0016910 | lld:lifeskim |
| pubmed-article:17118179 | pubmed:dateCreated | 2006-12-4 | lld:pubmed |
| pubmed-article:17118179 | pubmed:abstractText | Atovaquone is part of the antimalarial drug combination atovaquone-proguanil (Malarone) and inhibits the cytochrome bc1 complex of the electron transport chain in Plasmodium spp. Molecular modelling showed that amino acid mutations are clustered around a putative atovaquone-binding site resulting in a reduced binding affinity of atovaquone for plasmodial cytochrome b, thus resulting in drug resistance. | lld:pubmed |
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| pubmed-article:17118179 | pubmed:language | eng | lld:pubmed |
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| pubmed-article:17118179 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:17118179 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:17118179 | pubmed:issn | 1475-2875 | lld:pubmed |
| pubmed-article:17118179 | pubmed:author | pubmed-author:KunJürgen F... | lld:pubmed |
| pubmed-article:17118179 | pubmed:author | pubmed-author:KremsnerPeter... | lld:pubmed |
| pubmed-article:17118179 | pubmed:author | pubmed-author:HailuAsratA | lld:pubmed |
| pubmed-article:17118179 | pubmed:author | pubmed-author:GrobuschMarti... | lld:pubmed |
| pubmed-article:17118179 | pubmed:author | pubmed-author:GebruTamiratT | lld:pubmed |
| pubmed-article:17118179 | pubmed:issnType | Electronic | lld:pubmed |
| pubmed-article:17118179 | pubmed:volume | 5 | lld:pubmed |
| pubmed-article:17118179 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:17118179 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:17118179 | pubmed:pagination | 112 | lld:pubmed |
| pubmed-article:17118179 | pubmed:dateRevised | 2010-11-18 | lld:pubmed |
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| pubmed-article:17118179 | pubmed:meshHeading | pubmed-meshheading:17118179... | lld:pubmed |
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| pubmed-article:17118179 | pubmed:meshHeading | pubmed-meshheading:17118179... | lld:pubmed |
| pubmed-article:17118179 | pubmed:year | 2006 | lld:pubmed |
| pubmed-article:17118179 | pubmed:articleTitle | Molecular surveillance of mutations in the cytochrome b gene of Plasmodium falciparum in Gabon and Ethiopia. | lld:pubmed |
| pubmed-article:17118179 | pubmed:affiliation | Department of Medical Laboratory Technology, Faculty of Health Sciences, Haramaya University, Haramaya, Ethiopia. tamiratgw2002@yahoo.com <tamiratgw2002@yahoo.com> | lld:pubmed |
| pubmed-article:17118179 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:17118179 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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