| pubmed-article:1707931 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:1707931 | lifeskim:mentions | umls-concept:C0022688 | lld:lifeskim |
| pubmed-article:1707931 | lifeskim:mentions | umls-concept:C0022687 | lld:lifeskim |
| pubmed-article:1707931 | lifeskim:mentions | umls-concept:C0077503 | lld:lifeskim |
| pubmed-article:1707931 | lifeskim:mentions | umls-concept:C0441655 | lld:lifeskim |
| pubmed-article:1707931 | lifeskim:mentions | umls-concept:C1314939 | lld:lifeskim |
| pubmed-article:1707931 | lifeskim:mentions | umls-concept:C0079411 | lld:lifeskim |
| pubmed-article:1707931 | lifeskim:mentions | umls-concept:C1514485 | lld:lifeskim |
| pubmed-article:1707931 | pubmed:issue | 9 | lld:pubmed |
| pubmed-article:1707931 | pubmed:dateCreated | 1991-5-23 | lld:pubmed |
| pubmed-article:1707931 | pubmed:abstractText | In this study we investigated the expression of the 55 kDa (p55) and the 75 kDa (p75) TNF receptors in CD56+ NK cells and their role in NK and lymphokine-activated killer cells cell functions. By using mAb against the p55 and p75 TNF-R, NK cells were found to express both p55 and p75 upon activation, and both receptors were involved in the generation of lymphokine-activated killer cells activity. Proliferative activity of IL-2 stimulated NK cells was inhibited by anti-TNF-alpha mAb, indicating that endogenously produced TNF-alpha is important for optimal proliferation of NK cells. Furthermore, addition of rTNF-alpha increased the IL-2-induced proliferation of NK cells. mAb to p55 and p75 inhibited the IL-2-induced proliferation indicating that both TNF-R are involved in mediating this effect. | lld:pubmed |
| pubmed-article:1707931 | pubmed:language | eng | lld:pubmed |
| pubmed-article:1707931 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:1707931 | pubmed:citationSubset | AIM | lld:pubmed |
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| pubmed-article:1707931 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:1707931 | pubmed:month | May | lld:pubmed |
| pubmed-article:1707931 | pubmed:issn | 0022-1767 | lld:pubmed |
| pubmed-article:1707931 | pubmed:author | pubmed-author:LesslauerWW | lld:pubmed |
| pubmed-article:1707931 | pubmed:author | pubmed-author:EspevikTT | lld:pubmed |
| pubmed-article:1707931 | pubmed:author | pubmed-author:NaultRR | lld:pubmed |
| pubmed-article:1707931 | pubmed:author | pubmed-author:ShalabyRR | lld:pubmed |
| pubmed-article:1707931 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:1707931 | pubmed:day | 1 | lld:pubmed |
| pubmed-article:1707931 | pubmed:volume | 146 | lld:pubmed |
| pubmed-article:1707931 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:1707931 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:1707931 | pubmed:pagination | 3045-8 | lld:pubmed |
| pubmed-article:1707931 | pubmed:dateRevised | 2008-11-21 | lld:pubmed |
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| pubmed-article:1707931 | pubmed:year | 1991 | lld:pubmed |
| pubmed-article:1707931 | pubmed:articleTitle | Involvement of the 55- and 75-kDa tumor necrosis factor receptors in the generation of lymphokine-activated killer cell activity and proliferation of natural killer cells. | lld:pubmed |
| pubmed-article:1707931 | pubmed:affiliation | Institute of Cancer Research, University of Trondheim, Norway. | lld:pubmed |
| pubmed-article:1707931 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:1707931 | pubmed:publicationType | In Vitro | lld:pubmed |
| pubmed-article:1707931 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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