17060383

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Subject	Predicate	Object	Context
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pubmed-article:17060383	lifeskim:mentions	umls-concept:C0205263	lld:lifeskim
pubmed-article:17060383	pubmed:issue	18	lld:pubmed
pubmed-article:17060383	pubmed:dateCreated	2006-10-31	lld:pubmed
pubmed-article:17060383	pubmed:abstractText	Oxidation of low-density lipoprotein (LDL) and the subsequent processing of oxidized LDL (oxLDL) by macrophages results in activation of specific T cells, which contributes to the development of atherosclerosis. Oral tolerance induction and the subsequent activation of regulatory T cells may be an adequate therapy for the treatment of atherosclerosis.	lld:pubmed
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pubmed-article:17060383	pubmed:month	Oct	lld:pubmed
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pubmed-article:17060383	pubmed:author	pubmed-author:van der ZeeRR	lld:pubmed
pubmed-article:17060383	pubmed:author	pubmed-author:de VosPP	lld:pubmed
pubmed-article:17060383	pubmed:author	pubmed-author:KuiperJJ	lld:pubmed
pubmed-article:17060383	pubmed:author	pubmed-author:van EdenWW	lld:pubmed
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pubmed-article:17060383	pubmed:day	31	lld:pubmed
pubmed-article:17060383	pubmed:volume	114	lld:pubmed
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pubmed-article:17060383	pubmed:year	2006	lld:pubmed
pubmed-article:17060383	pubmed:articleTitle	Induction of oral tolerance to oxidized low-density lipoprotein ameliorates atherosclerosis.	lld:pubmed
pubmed-article:17060383	pubmed:affiliation	Division of Biopharmaceutics, Leiden University, Leiden, The Netherlands. puijvelde@lacdr.leidenuniv.nl	lld:pubmed
pubmed-article:17060383	pubmed:publicationType	Journal Article	lld:pubmed
pubmed-article:17060383	pubmed:publicationType	Research Support, Non-U.S. Gov't	lld:pubmed
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