pubmed-article:17052351 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:17052351 | lifeskim:mentions | umls-concept:C0030567 | lld:lifeskim |
pubmed-article:17052351 | lifeskim:mentions | umls-concept:C0038590 | lld:lifeskim |
pubmed-article:17052351 | lifeskim:mentions | umls-concept:C0009528 | lld:lifeskim |
pubmed-article:17052351 | lifeskim:mentions | umls-concept:C2603343 | lld:lifeskim |
pubmed-article:17052351 | pubmed:dateCreated | 2006-10-30 | lld:pubmed |
pubmed-article:17052351 | pubmed:abstractText | Inflammatory processes are increased in the Parkinson's disease (PD) brain. The long-term use of nonsteroidal anti-inflammatory drugs has been associated, in retrospective studies, with decreased risk for PD, suggesting that inflammation may contribute to development of this disorder. The objective of this study was to determine the extent of complement activation, a major inflammatory mechanism, in PD. | lld:pubmed |
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pubmed-article:17052351 | pubmed:language | eng | lld:pubmed |
pubmed-article:17052351 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:17052351 | pubmed:status | PubMed-not-MEDLINE | lld:pubmed |
pubmed-article:17052351 | pubmed:issn | 1742-2094 | lld:pubmed |
pubmed-article:17052351 | pubmed:author | pubmed-author:LoefflerDavid... | lld:pubmed |
pubmed-article:17052351 | pubmed:author | pubmed-author:ConantStephan... | lld:pubmed |
pubmed-article:17052351 | pubmed:author | pubmed-author:CampDianne... | lld:pubmed |
pubmed-article:17052351 | pubmed:issnType | Electronic | lld:pubmed |
pubmed-article:17052351 | pubmed:volume | 3 | lld:pubmed |
pubmed-article:17052351 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:17052351 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:17052351 | pubmed:pagination | 29 | lld:pubmed |
pubmed-article:17052351 | pubmed:dateRevised | 2009-11-18 | lld:pubmed |
pubmed-article:17052351 | pubmed:year | 2006 | lld:pubmed |
pubmed-article:17052351 | pubmed:articleTitle | Complement activation in the Parkinson's disease substantia nigra: an immunocytochemical study. | lld:pubmed |
pubmed-article:17052351 | pubmed:affiliation | Division of Neurology, William Beaumont Hospital Research Institute, Royal Oak, MI 48073, USA. DLoeffler@beaumont.edu | lld:pubmed |
pubmed-article:17052351 | pubmed:publicationType | Journal Article | lld:pubmed |
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