pubmed-article:17043167 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:17043167 | lifeskim:mentions | umls-concept:C0027051 | lld:lifeskim |
pubmed-article:17043167 | lifeskim:mentions | umls-concept:C0034693 | lld:lifeskim |
pubmed-article:17043167 | lifeskim:mentions | umls-concept:C0018787 | lld:lifeskim |
pubmed-article:17043167 | lifeskim:mentions | umls-concept:C0667931 | lld:lifeskim |
pubmed-article:17043167 | lifeskim:mentions | umls-concept:C0439659 | lld:lifeskim |
pubmed-article:17043167 | lifeskim:mentions | umls-concept:C1948023 | lld:lifeskim |
pubmed-article:17043167 | lifeskim:mentions | umls-concept:C0443252 | lld:lifeskim |
pubmed-article:17043167 | lifeskim:mentions | umls-concept:C1292733 | lld:lifeskim |
pubmed-article:17043167 | pubmed:issue | 18 | lld:pubmed |
pubmed-article:17043167 | pubmed:dateCreated | 2006-10-31 | lld:pubmed |
pubmed-article:17043167 | pubmed:abstractText | Adenosine inhibits proliferation of cardiac fibroblasts and hypertrophy of cardiomyocytes, both of which may play crucial roles in cardiac remodeling. In the present study, we investigated whether chronic stimulation of adenosine receptors begun after myocardial infarction (MI) prevents cardiac remodeling. | lld:pubmed |
pubmed-article:17043167 | pubmed:language | eng | lld:pubmed |
pubmed-article:17043167 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:17043167 | pubmed:citationSubset | AIM | lld:pubmed |
pubmed-article:17043167 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:17043167 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:17043167 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:17043167 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:17043167 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:17043167 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:17043167 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:17043167 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:17043167 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:17043167 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:17043167 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:17043167 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:17043167 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:17043167 | pubmed:month | Oct | lld:pubmed |
pubmed-article:17043167 | pubmed:issn | 1524-4539 | lld:pubmed |
pubmed-article:17043167 | pubmed:author | pubmed-author:KomamuraKazuo... | lld:pubmed |
pubmed-article:17043167 | pubmed:author | pubmed-author:KitakazeMasaf... | lld:pubmed |
pubmed-article:17043167 | pubmed:author | pubmed-author:TakashimaSeij... | lld:pubmed |
pubmed-article:17043167 | pubmed:author | pubmed-author:AsanumaHirosh... | lld:pubmed |
pubmed-article:17043167 | pubmed:author | pubmed-author:MochizukiNaok... | lld:pubmed |
pubmed-article:17043167 | pubmed:author | pubmed-author:MinaminoTetsu... | lld:pubmed |
pubmed-article:17043167 | pubmed:author | pubmed-author:KimJiyoongJ | lld:pubmed |
pubmed-article:17043167 | pubmed:author | pubmed-author:FujitaMasashi... | lld:pubmed |
pubmed-article:17043167 | pubmed:author | pubmed-author:SeguchiOsamuO | lld:pubmed |
pubmed-article:17043167 | pubmed:author | pubmed-author:OkadaKen-ichi... | lld:pubmed |
pubmed-article:17043167 | pubmed:author | pubmed-author:HirataAkioA | lld:pubmed |
pubmed-article:17043167 | pubmed:author | pubmed-author:TsukamotoOsam... | lld:pubmed |
pubmed-article:17043167 | pubmed:author | pubmed-author:WakenoMasakat... | lld:pubmed |
pubmed-article:17043167 | pubmed:author | pubmed-author:OkazakiHideto... | lld:pubmed |
pubmed-article:17043167 | pubmed:issnType | Electronic | lld:pubmed |
pubmed-article:17043167 | pubmed:day | 31 | lld:pubmed |
pubmed-article:17043167 | pubmed:volume | 114 | lld:pubmed |
pubmed-article:17043167 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:17043167 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:17043167 | pubmed:pagination | 1923-32 | lld:pubmed |
pubmed-article:17043167 | pubmed:dateRevised | 2010-11-18 | lld:pubmed |
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pubmed-article:17043167 | pubmed:year | 2006 | lld:pubmed |
pubmed-article:17043167 | pubmed:articleTitle | Long-term stimulation of adenosine A2b receptors begun after myocardial infarction prevents cardiac remodeling in rats. | lld:pubmed |
pubmed-article:17043167 | pubmed:affiliation | Department of Bioregulatory Medicine, Osaka University Graduate School of Medicine, Suita, Osaka, Japan. | lld:pubmed |
pubmed-article:17043167 | pubmed:publicationType | Journal Article | lld:pubmed |
pubmed-article:17043167 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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