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pubmed-article:16979603pubmed:abstractTextThe role of mitochondria and in particular of mitochondrial uncoupling in the mechanism of cardioprotection is not defined. In the accompanying paper we have shown that pretreatment of isolated rat hearts with a low concentration (100 nM) of FCCP, prior to global ischaemia, is cardioprotective, while 300 nM FCCP exacerbates injury. Here we define the mitochondrial responses to increasing concentrations of FCCP and also to explore the equivalence of the cardioprotective doses of diazoxide.lld:pubmed
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pubmed-article:16979603pubmed:articleTitleFCCP is cardioprotective at concentrations that cause mitochondrial oxidation without detectable depolarisation.lld:pubmed
pubmed-article:16979603pubmed:affiliationCardiac Physiology (Cardiovascular Division), King's College London, SE1 7EH, UK.lld:pubmed
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