| pubmed-article:16940123 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:16940123 | lifeskim:mentions | umls-concept:C0085484 | lld:lifeskim |
| pubmed-article:16940123 | lifeskim:mentions | umls-concept:C0032136 | lld:lifeskim |
| pubmed-article:16940123 | lifeskim:mentions | umls-concept:C0031437 | lld:lifeskim |
| pubmed-article:16940123 | lifeskim:mentions | umls-concept:C0242646 | lld:lifeskim |
| pubmed-article:16940123 | lifeskim:mentions | umls-concept:C0206243 | lld:lifeskim |
| pubmed-article:16940123 | lifeskim:mentions | umls-concept:C1515655 | lld:lifeskim |
| pubmed-article:16940123 | lifeskim:mentions | umls-concept:C0127400 | lld:lifeskim |
| pubmed-article:16940123 | pubmed:issue | 9 | lld:pubmed |
| pubmed-article:16940123 | pubmed:dateCreated | 2006-8-30 | lld:pubmed |
| pubmed-article:16940123 | pubmed:abstractText | Resistance to beta-lactams and quinolones in two isogenic Enterobacter cloacae isolates was studied. One was susceptible to cefoxitin and amoxicillin-clavulanate. The other one showed its natural beta-lactam resistance pattern. Both isolates had a nonfunctional AmpR regulator. However, within the second one, the presence of a plasmid carrying ampR and qnrA1 allowed reversion to the wild-type beta-lactam resistance phenotype and decreased susceptibility to fluoroquinolones. | lld:pubmed |
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| pubmed-article:16940123 | pubmed:language | eng | lld:pubmed |
| pubmed-article:16940123 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:16940123 | pubmed:citationSubset | IM | lld:pubmed |
| pubmed-article:16940123 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:16940123 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
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| pubmed-article:16940123 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:16940123 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:16940123 | pubmed:month | Sep | lld:pubmed |
| pubmed-article:16940123 | pubmed:issn | 0066-4804 | lld:pubmed |
| pubmed-article:16940123 | pubmed:author | pubmed-author:PrattFF | lld:pubmed |
| pubmed-article:16940123 | pubmed:author | pubmed-author:BartoloméR... | lld:pubmed |
| pubmed-article:16940123 | pubmed:author | pubmed-author:SabatéMM | lld:pubmed |
| pubmed-article:16940123 | pubmed:author | pubmed-author:González-Lópe... | lld:pubmed |
| pubmed-article:16940123 | pubmed:author | pubmed-author:LavillaSS | lld:pubmed |
| pubmed-article:16940123 | pubmed:author | pubmed-author:LarrosaM NMN | lld:pubmed |
| pubmed-article:16940123 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:16940123 | pubmed:volume | 50 | lld:pubmed |
| pubmed-article:16940123 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:16940123 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:16940123 | pubmed:pagination | 3175-8 | lld:pubmed |
| pubmed-article:16940123 | pubmed:dateRevised | 2009-11-18 | lld:pubmed |
| pubmed-article:16940123 | pubmed:meshHeading | pubmed-meshheading:16940123... | lld:pubmed |
| pubmed-article:16940123 | pubmed:meshHeading | pubmed-meshheading:16940123... | lld:pubmed |
| pubmed-article:16940123 | pubmed:meshHeading | pubmed-meshheading:16940123... | lld:pubmed |
| pubmed-article:16940123 | pubmed:meshHeading | pubmed-meshheading:16940123... | lld:pubmed |
| pubmed-article:16940123 | pubmed:meshHeading | pubmed-meshheading:16940123... | lld:pubmed |
| pubmed-article:16940123 | pubmed:meshHeading | pubmed-meshheading:16940123... | lld:pubmed |
| pubmed-article:16940123 | pubmed:meshHeading | pubmed-meshheading:16940123... | lld:pubmed |
| pubmed-article:16940123 | pubmed:meshHeading | pubmed-meshheading:16940123... | lld:pubmed |
| pubmed-article:16940123 | pubmed:meshHeading | pubmed-meshheading:16940123... | lld:pubmed |
| pubmed-article:16940123 | pubmed:meshHeading | pubmed-meshheading:16940123... | lld:pubmed |
| pubmed-article:16940123 | pubmed:meshHeading | pubmed-meshheading:16940123... | lld:pubmed |
| pubmed-article:16940123 | pubmed:year | 2006 | lld:pubmed |
| pubmed-article:16940123 | pubmed:articleTitle | In vivo reversion to the wild-type beta-lactam resistance phenotype mediated by a plasmid carrying ampR and qnrA1 in Enterobacter cloacae. | lld:pubmed |
| pubmed-article:16940123 | pubmed:affiliation | Servicio de Microbiología, Hospital Vall d'Hebron, Universitat Autònoma de Barcelona, Spain. | lld:pubmed |
| pubmed-article:16940123 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:16940123 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
