pubmed-article:16912055 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:16912055 | lifeskim:mentions | umls-concept:C0007226 | lld:lifeskim |
pubmed-article:16912055 | lifeskim:mentions | umls-concept:C0035647 | lld:lifeskim |
pubmed-article:16912055 | lifeskim:mentions | umls-concept:C0948265 | lld:lifeskim |
pubmed-article:16912055 | lifeskim:mentions | umls-concept:C0882849 | lld:lifeskim |
pubmed-article:16912055 | lifeskim:mentions | umls-concept:C0175630 | lld:lifeskim |
pubmed-article:16912055 | pubmed:issue | 18 | lld:pubmed |
pubmed-article:16912055 | pubmed:dateCreated | 2006-9-7 | lld:pubmed |
pubmed-article:16912055 | pubmed:abstractText | Circulating progenitor cells are believed to participate in cardiovascular (CV) homeostasis and their exhaustion has been linked to CV damage. As general agreement on their characterization is lacking, this work was carried out to assess the relationships between different antigenic profiles of progenitor cells and CV risk, with special regard to metabolic syndrome (MetSyn). | lld:pubmed |
pubmed-article:16912055 | pubmed:language | eng | lld:pubmed |
pubmed-article:16912055 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:16912055 | pubmed:citationSubset | IM | lld:pubmed |
pubmed-article:16912055 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:16912055 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:16912055 | pubmed:month | Sep | lld:pubmed |
pubmed-article:16912055 | pubmed:issn | 0195-668X | lld:pubmed |
pubmed-article:16912055 | pubmed:author | pubmed-author:TiengoAntonio... | lld:pubmed |
pubmed-article:16912055 | pubmed:author | pubmed-author:AgostiniCarlo... | lld:pubmed |
pubmed-article:16912055 | pubmed:author | pubmed-author:AvogaroAngelo... | lld:pubmed |
pubmed-article:16912055 | pubmed:author | pubmed-author:CoracinaAnnaA | lld:pubmed |
pubmed-article:16912055 | pubmed:author | pubmed-author:de... | lld:pubmed |
pubmed-article:16912055 | pubmed:author | pubmed-author:BaessoIleniaI | lld:pubmed |
pubmed-article:16912055 | pubmed:author | pubmed-author:FadiniGian... | lld:pubmed |
pubmed-article:16912055 | pubmed:issnType | Print | lld:pubmed |
pubmed-article:16912055 | pubmed:volume | 27 | lld:pubmed |
pubmed-article:16912055 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:16912055 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:16912055 | pubmed:pagination | 2247-55 | lld:pubmed |
pubmed-article:16912055 | pubmed:meshHeading | pubmed-meshheading:16912055... | lld:pubmed |
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pubmed-article:16912055 | pubmed:meshHeading | pubmed-meshheading:16912055... | lld:pubmed |
pubmed-article:16912055 | pubmed:meshHeading | pubmed-meshheading:16912055... | lld:pubmed |
pubmed-article:16912055 | pubmed:year | 2006 | lld:pubmed |
pubmed-article:16912055 | pubmed:articleTitle | Circulating CD34+ cells, metabolic syndrome, and cardiovascular risk. | lld:pubmed |
pubmed-article:16912055 | pubmed:affiliation | Department of Clinical and Experimental Medicine, Division of Metabolic Diseases, University of Padua, Medical School, Padua, Italy. | lld:pubmed |
pubmed-article:16912055 | pubmed:publicationType | Journal Article | lld:pubmed |
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