| pubmed-article:16877415 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:16877415 | lifeskim:mentions | umls-concept:C1112768 | lld:lifeskim |
| pubmed-article:16877415 | lifeskim:mentions | umls-concept:C0040690 | lld:lifeskim |
| pubmed-article:16877415 | lifeskim:mentions | umls-concept:C0332197 | lld:lifeskim |
| pubmed-article:16877415 | lifeskim:mentions | umls-concept:C0017262 | lld:lifeskim |
| pubmed-article:16877415 | lifeskim:mentions | umls-concept:C0919432 | lld:lifeskim |
| pubmed-article:16877415 | lifeskim:mentions | umls-concept:C0205263 | lld:lifeskim |
| pubmed-article:16877415 | lifeskim:mentions | umls-concept:C1522492 | lld:lifeskim |
| pubmed-article:16877415 | lifeskim:mentions | umls-concept:C2911684 | lld:lifeskim |
| pubmed-article:16877415 | lifeskim:mentions | umls-concept:C0185117 | lld:lifeskim |
| pubmed-article:16877415 | pubmed:issue | 8 | lld:pubmed |
| pubmed-article:16877415 | pubmed:dateCreated | 2006-7-31 | lld:pubmed |
| pubmed-article:16877415 | pubmed:abstractText | Smad3, a mediator of TGF-beta signaling has been shown to be involved in the epithelial-to-mesenchymal transformation (EMT) of lens epithelial cells in a lens injury model. In this study, the role of Smad3 in anterior subcapsular cataract (ASC) formation was investigated in a transgenic TGF-beta/Smad3 knockout mouse model. | lld:pubmed |
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| pubmed-article:16877415 | pubmed:language | eng | lld:pubmed |
| pubmed-article:16877415 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:16877415 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:16877415 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:16877415 | pubmed:month | Aug | lld:pubmed |
| pubmed-article:16877415 | pubmed:issn | 0146-0404 | lld:pubmed |
| pubmed-article:16877415 | pubmed:author | pubmed-author:SivakJacob... | lld:pubmed |
