pubmed-article:16847436 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:16847436 | lifeskim:mentions | umls-concept:C0037925 | lld:lifeskim |
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pubmed-article:16847436 | lifeskim:mentions | umls-concept:C0040845 | lld:lifeskim |
pubmed-article:16847436 | lifeskim:mentions | umls-concept:C0042839 | lld:lifeskim |
pubmed-article:16847436 | lifeskim:mentions | umls-concept:C0205177 | lld:lifeskim |
pubmed-article:16847436 | lifeskim:mentions | umls-concept:C1280500 | lld:lifeskim |
pubmed-article:16847436 | lifeskim:mentions | umls-concept:C1325847 | lld:lifeskim |
pubmed-article:16847436 | lifeskim:mentions | umls-concept:C0870883 | lld:lifeskim |
pubmed-article:16847436 | lifeskim:mentions | umls-concept:C0205263 | lld:lifeskim |
pubmed-article:16847436 | pubmed:issue | 1 | lld:pubmed |
pubmed-article:16847436 | pubmed:dateCreated | 2006-8-28 | lld:pubmed |
pubmed-article:16847436 | pubmed:abstractText | Retinoic acid is an active metabolite of vitamin A involved in the modulation of the inflammatory and nociceptive responses. The aim of the present study was to analyze the properties of spinal cord neuronal responses of male Wistar rats treated with all-trans retinoic acid (ATRA) p.o. in the normal situation and under carrageenan-induced inflammation. We also studied the expression and distribution of cyclooxygenases (COX) in the spinal cord. | lld:pubmed |
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pubmed-article:16847436 | pubmed:language | eng | lld:pubmed |
pubmed-article:16847436 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:16847436 | pubmed:citationSubset | IM | lld:pubmed |
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pubmed-article:16847436 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:16847436 | pubmed:month | Sep | lld:pubmed |
pubmed-article:16847436 | pubmed:issn | 0007-1188 | lld:pubmed |
pubmed-article:16847436 | pubmed:author | pubmed-author:MolinaCC | lld:pubmed |
pubmed-article:16847436 | pubmed:author | pubmed-author:HerreroJ FJF | lld:pubmed |
pubmed-article:16847436 | pubmed:author | pubmed-author:LucioF JFJ | lld:pubmed |
pubmed-article:16847436 | pubmed:author | pubmed-author:AliquoCC | lld:pubmed |
pubmed-article:16847436 | pubmed:author | pubmed-author:Moreno-Manzan... | lld:pubmed |
pubmed-article:16847436 | pubmed:author | pubmed-author:Romero-Sandov... | lld:pubmed |
pubmed-article:16847436 | pubmed:issnType | Print | lld:pubmed |
pubmed-article:16847436 | pubmed:volume | 149 | lld:pubmed |
pubmed-article:16847436 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:16847436 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:16847436 | pubmed:pagination | 56-64 | lld:pubmed |
pubmed-article:16847436 | pubmed:dateRevised | 2009-11-18 | lld:pubmed |
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pubmed-article:16847436 | pubmed:meshHeading | pubmed-meshheading:16847436... | lld:pubmed |
pubmed-article:16847436 | pubmed:year | 2006 | lld:pubmed |
pubmed-article:16847436 | pubmed:articleTitle | Vitamin A active metabolite, all-trans retinoic acid, induces spinal cord sensitization. I. Effects after oral administration. | lld:pubmed |
pubmed-article:16847436 | pubmed:affiliation | Departamento de Fisiología, Facultad de Medicina, Universidad de Alcalá, Madrid, Spain. | lld:pubmed |
pubmed-article:16847436 | pubmed:publicationType | Journal Article | lld:pubmed |
pubmed-article:16847436 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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