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pubmed-article:16829721pubmed:abstractTextPharmacological interventions may play a prominent role in reducing organ damage in response to physiologic stress. A growing body of evidence indicates that volatile anesthetics exert protective effects against ischemia-reperfusion injury in vivo. Administration of volatile anesthetics before prolonged coronary artery occlusion and reperfusion has been shown to produce cardioprotection, a phenomenon termed anesthetic-induced preconditioning. Endogenous signal transduction proteins, reactive oxygen species, mitochondria, and ion channels have been implicated in anesthetic-induced preconditioning, and new data regarding the triggering and effector roles for these various components have been discovered that advance our understanding of the mechanisms responsible for anesthetic-induced preconditioning. This review will update and integrate these recent data into the current mechanistic model of anesthetic-induced preconditioning.lld:pubmed
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pubmed-article:16829721pubmed:pagination397-403lld:pubmed
pubmed-article:16829721pubmed:dateRevised2011-11-17lld:pubmed
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pubmed-article:16829721pubmed:articleTitleCardioprotection by volatile anesthetics: new applications for old drugs?lld:pubmed
pubmed-article:16829721pubmed:affiliationDepartment of Anesthesiology, Medical College of Wisconsin, Milwaukee, Wisconsin 53226, USA.lld:pubmed
pubmed-article:16829721pubmed:publicationTypeJournal Articlelld:pubmed
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