pubmed-article:16735799 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:16735799 | lifeskim:mentions | umls-concept:C0026809 | lld:lifeskim |
pubmed-article:16735799 | lifeskim:mentions | umls-concept:C0022646 | lld:lifeskim |
pubmed-article:16735799 | lifeskim:mentions | umls-concept:C2750067 | lld:lifeskim |
pubmed-article:16735799 | lifeskim:mentions | umls-concept:C0814225 | lld:lifeskim |
pubmed-article:16735799 | lifeskim:mentions | umls-concept:C0205373 | lld:lifeskim |
pubmed-article:16735799 | lifeskim:mentions | umls-concept:C0686907 | lld:lifeskim |
pubmed-article:16735799 | pubmed:issue | 1 | lld:pubmed |
pubmed-article:16735799 | pubmed:dateCreated | 2006-8-16 | lld:pubmed |
pubmed-article:16735799 | pubmed:abstractText | Elevated plasma levels of plasminogen activator inhibitor-1 (PAI-1) are observed in patients with obesity, hypertension and diabetes, and several observations suggest that PAI-1 mediates diabetic vascular complications. Although increased intrarenal expression of PAI-1 is also a feature of diabetic nephropathy, evidence that PAI-1 plays a primary pathogenetic role in the renal pathology is lacking. | lld:pubmed |
pubmed-article:16735799 | pubmed:grant | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:16735799 | pubmed:language | eng | lld:pubmed |
pubmed-article:16735799 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:16735799 | pubmed:citationSubset | IM | lld:pubmed |
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pubmed-article:16735799 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:16735799 | pubmed:issn | 1660-2129 | lld:pubmed |
pubmed-article:16735799 | pubmed:author | pubmed-author:EddyAllison... | lld:pubmed |
pubmed-article:16735799 | pubmed:author | pubmed-author:ZhangGuoqiang... | lld:pubmed |
pubmed-article:16735799 | pubmed:author | pubmed-author:CaiXiaoheX | lld:pubmed |
pubmed-article:16735799 | pubmed:author | pubmed-author:Lopez-GuisaJe... | lld:pubmed |
pubmed-article:16735799 | pubmed:author | pubmed-author:MatsuoShunyaS | lld:pubmed |
pubmed-article:16735799 | pubmed:author | pubmed-author:ChuaStreamson... | lld:pubmed |
pubmed-article:16735799 | pubmed:author | pubmed-author:CollinsSarah... | lld:pubmed |
pubmed-article:16735799 | pubmed:author | pubmed-author:MaruvadaRaviR | lld:pubmed |
pubmed-article:16735799 | pubmed:author | pubmed-author:OkamuraDaryl... | lld:pubmed |
pubmed-article:16735799 | pubmed:author | pubmed-author:AlexanderShan... | lld:pubmed |
pubmed-article:16735799 | pubmed:copyrightInfo | Copyright 2006 S. Karger AG, Basel. | lld:pubmed |
pubmed-article:16735799 | pubmed:issnType | Electronic | lld:pubmed |
pubmed-article:16735799 | pubmed:volume | 104 | lld:pubmed |
pubmed-article:16735799 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:16735799 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:16735799 | pubmed:pagination | e23-34 | lld:pubmed |
pubmed-article:16735799 | pubmed:dateRevised | 2008-11-21 | lld:pubmed |
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pubmed-article:16735799 | pubmed:year | 2006 | lld:pubmed |
pubmed-article:16735799 | pubmed:articleTitle | Plasminogen activator inhibitor-1 deficiency has renal benefits but some adverse systemic consequences in diabetic mice. | lld:pubmed |
pubmed-article:16735799 | pubmed:affiliation | Children's Hospital and Regional Medical Center, Department of Pediatrics, University of Washington, Seattle, Wash, USA. | lld:pubmed |
pubmed-article:16735799 | pubmed:publicationType | Journal Article | lld:pubmed |
pubmed-article:16735799 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
pubmed-article:16735799 | pubmed:publicationType | Research Support, N.I.H., Extramural | lld:pubmed |
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