pubmed-article:16708128 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:16708128 | lifeskim:mentions | umls-concept:C0678803 | lld:lifeskim |
pubmed-article:16708128 | lifeskim:mentions | umls-concept:C0038454 | lld:lifeskim |
pubmed-article:16708128 | lifeskim:mentions | umls-concept:C0162596 | lld:lifeskim |
pubmed-article:16708128 | lifeskim:mentions | umls-concept:C0442805 | lld:lifeskim |
pubmed-article:16708128 | lifeskim:mentions | umls-concept:C0004083 | lld:lifeskim |
pubmed-article:16708128 | lifeskim:mentions | umls-concept:C0205191 | lld:lifeskim |
pubmed-article:16708128 | pubmed:issue | 2 | lld:pubmed |
pubmed-article:16708128 | pubmed:dateCreated | 2006-5-18 | lld:pubmed |
pubmed-article:16708128 | pubmed:abstractText | A chemical worker working with urea-formaldehyde resin hazard for 20 years suffered cerebral ischemia in association with an increase of blood beta2-glycoprotein I-dependent anticardiolipin antibody (aCL)-IgG and IgM isotype, and a prolongation of activated partial thromboplastin time (aPTT). Major histocompatibility complex antigen showed DR4 positivity. On follow-up for over 6 years, aCL-IgG and aPTT decreased to reference range but aCL-IgM was still abnormally high despite a cessation of exposure. This patient highlights the induction of antibody-mediated thrombosis in chronic chemical exposure, especially in an individual with subclinical autoimmune disorder. The role of environment for coagulopathic vascular thrombosis is warranted for investigation. | lld:pubmed |
pubmed-article:16708128 | pubmed:language | eng | lld:pubmed |
pubmed-article:16708128 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:16708128 | pubmed:citationSubset | IM | lld:pubmed |
pubmed-article:16708128 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:16708128 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:16708128 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:16708128 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:16708128 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:16708128 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:16708128 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:16708128 | pubmed:month | Apr | lld:pubmed |
pubmed-article:16708128 | pubmed:issn | 1076-0296 | lld:pubmed |
pubmed-article:16708128 | pubmed:author | pubmed-author:LuiChun-Chung... | lld:pubmed |
pubmed-article:16708128 | pubmed:author | pubmed-author:ChenChung-Jen... | lld:pubmed |
pubmed-article:16708128 | pubmed:author | pubmed-author:ChenWei-HsiWH | lld:pubmed |
pubmed-article:16708128 | pubmed:author | pubmed-author:LiuJia-ShouJS | lld:pubmed |
pubmed-article:16708128 | pubmed:issnType | Print | lld:pubmed |
pubmed-article:16708128 | pubmed:volume | 12 | lld:pubmed |
pubmed-article:16708128 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:16708128 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:16708128 | pubmed:pagination | 231-5 | lld:pubmed |
pubmed-article:16708128 | pubmed:dateRevised | 2007-11-15 | lld:pubmed |
pubmed-article:16708128 | pubmed:meshHeading | pubmed-meshheading:16708128... | lld:pubmed |
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pubmed-article:16708128 | pubmed:meshHeading | pubmed-meshheading:16708128... | lld:pubmed |
pubmed-article:16708128 | pubmed:year | 2006 | lld:pubmed |
pubmed-article:16708128 | pubmed:articleTitle | An increase of anticardiolipin antibody in association with stroke and chronic chemical exposure. | lld:pubmed |
pubmed-article:16708128 | pubmed:affiliation | Stroke Biology Research Laboratory, Chang Gung Memorial Hospital, Kaohsiung, Taiwan. | lld:pubmed |
pubmed-article:16708128 | pubmed:publicationType | Journal Article | lld:pubmed |
pubmed-article:16708128 | pubmed:publicationType | Case Reports | lld:pubmed |