pubmed-article:16678699 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:16678699 | lifeskim:mentions | umls-concept:C0020507 | lld:lifeskim |
pubmed-article:16678699 | lifeskim:mentions | umls-concept:C1533698 | lld:lifeskim |
pubmed-article:16678699 | lifeskim:mentions | umls-concept:C0032611 | lld:lifeskim |
pubmed-article:16678699 | lifeskim:mentions | umls-concept:C0205332 | lld:lifeskim |
pubmed-article:16678699 | lifeskim:mentions | umls-concept:C0741847 | lld:lifeskim |
pubmed-article:16678699 | lifeskim:mentions | umls-concept:C1517499 | lld:lifeskim |
pubmed-article:16678699 | lifeskim:mentions | umls-concept:C1527362 | lld:lifeskim |
pubmed-article:16678699 | pubmed:issue | 5 | lld:pubmed |
pubmed-article:16678699 | pubmed:dateCreated | 2006-5-8 | lld:pubmed |
pubmed-article:16678699 | pubmed:abstractText | Intimal hyperplasia (IH) is most commonly the cause of graft occlusion in infrainguinal bypass grafting for arterial occlusive disease. We investigated the influence of nitric oxide on the IH of the arterial vessel wall at the region of prosthetic bypass anastomoses. | lld:pubmed |
pubmed-article:16678699 | pubmed:language | eng | lld:pubmed |
pubmed-article:16678699 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:16678699 | pubmed:citationSubset | IM | lld:pubmed |
pubmed-article:16678699 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:16678699 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:16678699 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:16678699 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:16678699 | pubmed:month | May | lld:pubmed |
pubmed-article:16678699 | pubmed:issn | 0741-5214 | lld:pubmed |
pubmed-article:16678699 | pubmed:author | pubmed-author:GödeckeAxelA | lld:pubmed |
pubmed-article:16678699 | pubmed:author | pubmed-author:SchraderJürge... | lld:pubmed |
pubmed-article:16678699 | pubmed:author | pubmed-author:SandmannWilhe... | lld:pubmed |
pubmed-article:16678699 | pubmed:author | pubmed-author:PfeifferTomas... | lld:pubmed |
pubmed-article:16678699 | pubmed:author | pubmed-author:WallichMartin... | lld:pubmed |
pubmed-article:16678699 | pubmed:issnType | Print | lld:pubmed |
pubmed-article:16678699 | pubmed:volume | 43 | lld:pubmed |
pubmed-article:16678699 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:16678699 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:16678699 | pubmed:pagination | 1021-7 | lld:pubmed |
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pubmed-article:16678699 | pubmed:meshHeading | pubmed-meshheading:16678699... | lld:pubmed |
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pubmed-article:16678699 | pubmed:meshHeading | pubmed-meshheading:16678699... | lld:pubmed |
pubmed-article:16678699 | pubmed:year | 2006 | lld:pubmed |
pubmed-article:16678699 | pubmed:articleTitle | Lipoplex gene transfer of inducible nitric oxide synthase inhibits the reactive intimal hyperplasia after expanded polytetrafluoroethylene bypass grafting. | lld:pubmed |
pubmed-article:16678699 | pubmed:affiliation | Department of Vascular Surgery and Kidney Transplantation, University Hospital Medical School, Heinrich-Heine-University Düsseldorf, Germany. tomas.pfeiffer@hbh-kliniken.de | lld:pubmed |
pubmed-article:16678699 | pubmed:publicationType | Journal Article | lld:pubmed |
http://linkedlifedata.com/r... | pubmed:referesTo | pubmed-article:16678699 | lld:pubmed |