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| pubmed-article:16636176 | lifeskim:mentions | umls-concept:C1521761 | lld:lifeskim |
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| pubmed-article:16636176 | lifeskim:mentions | umls-concept:C0392747 | lld:lifeskim |
| pubmed-article:16636176 | lifeskim:mentions | umls-concept:C0678594 | lld:lifeskim |
| pubmed-article:16636176 | lifeskim:mentions | umls-concept:C0205210 | lld:lifeskim |
| pubmed-article:16636176 | lifeskim:mentions | umls-concept:C1280464 | lld:lifeskim |
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| pubmed-article:16636176 | lifeskim:mentions | umls-concept:C0331858 | lld:lifeskim |
| pubmed-article:16636176 | pubmed:issue | 17 | lld:pubmed |
| pubmed-article:16636176 | pubmed:dateCreated | 2006-5-2 | lld:pubmed |
| pubmed-article:16636176 | pubmed:abstractText | Chronic hypertension may cause left ventricular (LV) remodeling, alterations in cardiac function, and the development of chronic heart failure (CHF). Changes in the composition of the extracellular matrix (ECM) known to occur in hypertension are believed to be causally related to these structural, functional, and clinical outcomes. However, whether the determinants of ECM composition, such as the balance between ECM proteases (matrix metalloproteinases [MMPs]) and their tissue inhibitors [TIMPs]), are altered in hypertensive heart disease is unknown. | lld:pubmed |
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| pubmed-article:16636176 | pubmed:language | eng | lld:pubmed |
| pubmed-article:16636176 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:16636176 | pubmed:citationSubset | AIM | lld:pubmed |
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| pubmed-article:16636176 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:16636176 | pubmed:month | May | lld:pubmed |
| pubmed-article:16636176 | pubmed:issn | 1524-4539 | lld:pubmed |
| pubmed-article:16636176 | pubmed:author | pubmed-author:SpinaleFranci... | lld:pubmed |
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| pubmed-article:16636176 | pubmed:author | pubmed-author:LeonardiAmy... | lld:pubmed |
| pubmed-article:16636176 | pubmed:author | pubmed-author:PenningtonWee... | lld:pubmed |
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| pubmed-article:16636176 | pubmed:author | pubmed-author:McClureCather... | lld:pubmed |
| pubmed-article:16636176 | pubmed:issnType | Electronic | lld:pubmed |
| pubmed-article:16636176 | pubmed:day | 2 | lld:pubmed |
| pubmed-article:16636176 | pubmed:volume | 113 | lld:pubmed |
| pubmed-article:16636176 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:16636176 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:16636176 | pubmed:pagination | 2089-96 | lld:pubmed |
| pubmed-article:16636176 | pubmed:dateRevised | 2007-11-15 | lld:pubmed |
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| pubmed-article:16636176 | pubmed:year | 2006 | lld:pubmed |
| pubmed-article:16636176 | pubmed:articleTitle | Matrix metalloproteinases/tissue inhibitors of metalloproteinases: relationship between changes in proteolytic determinants of matrix composition and structural, functional, and clinical manifestations of hypertensive heart disease. | lld:pubmed |
| pubmed-article:16636176 | pubmed:affiliation | Department of Medicine, Medical University of South Carolina, RHJ Department of Veterans Affairs Medical Center, Charleston, SC, USA. | lld:pubmed |
| pubmed-article:16636176 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:16636176 | pubmed:publicationType | Research Support, U.S. Gov't, Non-P.H.S. | lld:pubmed |
| pubmed-article:16636176 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
| pubmed-article:16636176 | pubmed:publicationType | Research Support, N.I.H., Extramural | lld:pubmed |
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