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pubmed-article:16557920pubmed:abstractTextAnti-rabbit alveolar macrophage goat serum (AMS), adsorbed of hemolysins and hemagglutinins, exhibited complement-dependent cytotoxicity for the following rabbit target cells: alveolar macrophages, thymocytes, and kidney (RK-13) cells. The intravenous injection of AMS, sufficient to reduce markedly the dermal tuberculin reaction in BCG-sensitized rabbits, did not suppress the development of BCG-induced chronic and accelerated pulmonary granulomas. These observations are discussed in relation to delayed sensitivity, allergic granuloma formation, and cell-associated immunity.lld:pubmed
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pubmed-article:16557920pubmed:authorpubmed-author:MyrvikQ NQNlld:pubmed
pubmed-article:16557920pubmed:authorpubmed-author:MooreV LVLlld:pubmed
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pubmed-article:16557920pubmed:pagination810-4lld:pubmed
pubmed-article:16557920pubmed:dateRevised2010-9-15lld:pubmed
pubmed-article:16557920pubmed:year1970lld:pubmed
pubmed-article:16557920pubmed:articleTitleEffect of antimacrophage serum on dermal tuberculin sensitivity and allergic pulmonary granuloma formation in rabbits.lld:pubmed
pubmed-article:16557920pubmed:affiliationDepartment of Microbiology, Bowman Gray School of Medicine of Wake Forest University, Winston-Salem, North Carolina 27103.lld:pubmed
pubmed-article:16557920pubmed:publicationTypeJournal Articlelld:pubmed
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