| pubmed-article:16481471 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:16481471 | lifeskim:mentions | umls-concept:C0079427 | lld:lifeskim |
| pubmed-article:16481471 | lifeskim:mentions | umls-concept:C0542341 | lld:lifeskim |
| pubmed-article:16481471 | lifeskim:mentions | umls-concept:C0694888 | lld:lifeskim |
| pubmed-article:16481471 | lifeskim:mentions | umls-concept:C0314603 | lld:lifeskim |
| pubmed-article:16481471 | lifeskim:mentions | umls-concept:C1527148 | lld:lifeskim |
| pubmed-article:16481471 | lifeskim:mentions | umls-concept:C0162610 | lld:lifeskim |
| pubmed-article:16481471 | lifeskim:mentions | umls-concept:C0024861 | lld:lifeskim |
| pubmed-article:16481471 | lifeskim:mentions | umls-concept:C1880371 | lld:lifeskim |
| pubmed-article:16481471 | lifeskim:mentions | umls-concept:C1313915 | lld:lifeskim |
| pubmed-article:16481471 | pubmed:issue | 4 | lld:pubmed |
| pubmed-article:16481471 | pubmed:dateCreated | 2006-2-16 | lld:pubmed |
| pubmed-article:16481471 | pubmed:abstractText | Genetic redundancy is associated with a large percentage of genes. We investigated PTEN (phosphatase and tensin homolog deleted on chromosome 10) tumor suppressor gene functions that eluded single mutant analyses, using a Caenorhabditis elegans genome-wide screen. We show that at least 27 genes collaborate with the worm PTEN homolog daf-18 for various functions previously concealed by genetic redundancy, including embryogenesis, cuticle turnover, egg laying, and oocyte maturation. In one example, daf-18 appears to constitute a cell-autonomous germline signal that converges with a somatic gonad signal mediated by ceh-18 at a kinase inhibition. We provide evidence that daf-18 elicits some functions independent of the downstream gene daf-16. | lld:pubmed |
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| pubmed-article:16481471 | pubmed:language | eng | lld:pubmed |
| pubmed-article:16481471 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:16481471 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:16481471 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:16481471 | pubmed:month | Feb | lld:pubmed |
| pubmed-article:16481471 | pubmed:issn | 0890-9369 | lld:pubmed |
| pubmed-article:16481471 | pubmed:author | pubmed-author:MeiJ LJL | lld:pubmed |
| pubmed-article:16481471 | pubmed:author | pubmed-author:SuzukiYoY | lld:pubmed |
| pubmed-article:16481471 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:16481471 | pubmed:day | 15 | lld:pubmed |
| pubmed-article:16481471 | pubmed:volume | 20 | lld:pubmed |
| pubmed-article:16481471 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:16481471 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:16481471 | pubmed:pagination | 423-8 | lld:pubmed |
| pubmed-article:16481471 | pubmed:dateRevised | 2009-11-19 | lld:pubmed |
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| pubmed-article:16481471 | pubmed:meshHeading | pubmed-meshheading:16481471... | lld:pubmed |
| pubmed-article:16481471 | pubmed:year | 2006 | lld:pubmed |
| pubmed-article:16481471 | pubmed:articleTitle | Genetic redundancy masks diverse functions of the tumor suppressor gene PTEN during C. elegans development. | lld:pubmed |
| pubmed-article:16481471 | pubmed:affiliation | Howard Hughes Medical Institute and Department of Molecular, Cellular, and Developmental Biology, University of Colorado, Boulder, Colorado 80309, USA. | lld:pubmed |
| pubmed-article:16481471 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:16481471 | pubmed:publicationType | Comparative Study | lld:pubmed |
| pubmed-article:16481471 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
| entrez-gene:172771 | entrezgene:pubmed | pubmed-article:16481471 | lld:entrezgene |
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