pubmed-article:16421589 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:16421589 | lifeskim:mentions | umls-concept:C1136215 | lld:lifeskim |
pubmed-article:16421589 | lifeskim:mentions | umls-concept:C1538577 | lld:lifeskim |
pubmed-article:16421589 | lifeskim:mentions | umls-concept:C0920425 | lld:lifeskim |
pubmed-article:16421589 | lifeskim:mentions | umls-concept:C1521840 | lld:lifeskim |
pubmed-article:16421589 | pubmed:issue | 3 | lld:pubmed |
pubmed-article:16421589 | pubmed:dateCreated | 2006-2-8 | lld:pubmed |
pubmed-article:16421589 | pubmed:abstractText | The requirement for the maintenance of telomeres by telomerase by most cancer cells for continued proliferation is a target in anticancer strategies. Tankyrases are poly(ADP-ribose) polymerases that enhance telomerase access to telomeres. Tankyrase 1 modulates telomerase inhibition in human cancer cells and is reviewed in this report as a potential telomere-directed anticancer target. | lld:pubmed |
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pubmed-article:16421589 | pubmed:language | eng | lld:pubmed |
pubmed-article:16421589 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:16421589 | pubmed:citationSubset | IM | lld:pubmed |
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pubmed-article:16421589 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:16421589 | pubmed:month | Feb | lld:pubmed |
pubmed-article:16421589 | pubmed:issn | 0007-0920 | lld:pubmed |
pubmed-article:16421589 | pubmed:author | pubmed-author:SeimiyaHH | lld:pubmed |
pubmed-article:16421589 | pubmed:issnType | Print | lld:pubmed |
pubmed-article:16421589 | pubmed:day | 13 | lld:pubmed |
pubmed-article:16421589 | pubmed:volume | 94 | lld:pubmed |
pubmed-article:16421589 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:16421589 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:16421589 | pubmed:pagination | 341-5 | lld:pubmed |
pubmed-article:16421589 | pubmed:dateRevised | 2009-11-18 | lld:pubmed |
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pubmed-article:16421589 | pubmed:year | 2006 | lld:pubmed |
pubmed-article:16421589 | pubmed:articleTitle | The telomeric PARP, tankyrases, as targets for cancer therapy. | lld:pubmed |
pubmed-article:16421589 | pubmed:affiliation | Division of Molecular Biotherapy, Cancer Chemotherapy Center, Japanese Foundation for Cancer Research, 3-10-6 Ariake, Koto-ku, Tokyo 135-8550, Japan. hseimiya@jfcr.or.jp | lld:pubmed |
pubmed-article:16421589 | pubmed:publicationType | Journal Article | lld:pubmed |
pubmed-article:16421589 | pubmed:publicationType | Review | lld:pubmed |
pubmed-article:16421589 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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