pubmed-article:16401840 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:16401840 | lifeskim:mentions | umls-concept:C0032105 | lld:lifeskim |
pubmed-article:16401840 | lifeskim:mentions | umls-concept:C0085220 | lld:lifeskim |
pubmed-article:16401840 | lifeskim:mentions | umls-concept:C2752009 | lld:lifeskim |
pubmed-article:16401840 | lifeskim:mentions | umls-concept:C0560005 | lld:lifeskim |
pubmed-article:16401840 | lifeskim:mentions | umls-concept:C2353566 | lld:lifeskim |
pubmed-article:16401840 | pubmed:issue | 1 | lld:pubmed |
pubmed-article:16401840 | pubmed:dateCreated | 2006-1-10 | lld:pubmed |
pubmed-article:16401840 | pubmed:abstractText | Microvascular brain injury, typically measured by extent of white matter hyperintensity (WMH) on MRI, is an important contributor to cognitive impairment in the elderly. Recent studies suggest a role for circulating beta-amyloid peptide in microvascular dysfunction and white matter disease. | lld:pubmed |
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pubmed-article:16401840 | pubmed:language | eng | lld:pubmed |
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pubmed-article:16401840 | pubmed:citationSubset | AIM | lld:pubmed |
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pubmed-article:16401840 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:16401840 | pubmed:month | Jan | lld:pubmed |
pubmed-article:16401840 | pubmed:issn | 1526-632X | lld:pubmed |
pubmed-article:16401840 | pubmed:author | pubmed-author:SmithE EEE | lld:pubmed |
pubmed-article:16401840 | pubmed:author | pubmed-author:GrowdonJ HJH | lld:pubmed |
pubmed-article:16401840 | pubmed:author | pubmed-author:RajuSS | lld:pubmed |
pubmed-article:16401840 | pubmed:author | pubmed-author:GreenbergS... | lld:pubmed |
pubmed-article:16401840 | pubmed:author | pubmed-author:BottiglieriTT | lld:pubmed |
pubmed-article:16401840 | pubmed:author | pubmed-author:Diaz-Arrastia... | lld:pubmed |
pubmed-article:16401840 | pubmed:author | pubmed-author:GuinnE CEC | lld:pubmed |
pubmed-article:16401840 | pubmed:author | pubmed-author:RosanoGG | lld:pubmed |
pubmed-article:16401840 | pubmed:author | pubmed-author:IrizarryM CMC | lld:pubmed |
pubmed-article:16401840 | pubmed:issnType | Electronic | lld:pubmed |
pubmed-article:16401840 | pubmed:day | 10 | lld:pubmed |
pubmed-article:16401840 | pubmed:volume | 66 | lld:pubmed |
pubmed-article:16401840 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:16401840 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:16401840 | pubmed:pagination | 23-9 | lld:pubmed |
pubmed-article:16401840 | pubmed:dateRevised | 2010-11-18 | lld:pubmed |
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pubmed-article:16401840 | pubmed:year | 2006 | lld:pubmed |
pubmed-article:16401840 | pubmed:articleTitle | Plasma beta-amyloid and white matter lesions in AD, MCI, and cerebral amyloid angiopathy. | lld:pubmed |
pubmed-article:16401840 | pubmed:affiliation | Department of Neurology, Massachusetts General Hospital, Boston, MA 02114, USA. | lld:pubmed |
pubmed-article:16401840 | pubmed:publicationType | Journal Article | lld:pubmed |
pubmed-article:16401840 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
pubmed-article:16401840 | pubmed:publicationType | Research Support, N.I.H., Extramural | lld:pubmed |
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