| pubmed-article:16390934 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:16390934 | lifeskim:mentions | umls-concept:C0020792 | lld:lifeskim |
| pubmed-article:16390934 | lifeskim:mentions | umls-concept:C0004359 | lld:lifeskim |
| pubmed-article:16390934 | lifeskim:mentions | umls-concept:C0035126 | lld:lifeskim |
| pubmed-article:16390934 | lifeskim:mentions | umls-concept:C1521840 | lld:lifeskim |
| pubmed-article:16390934 | pubmed:issue | 1 | lld:pubmed |
| pubmed-article:16390934 | pubmed:dateCreated | 2006-1-24 | lld:pubmed |
| pubmed-article:16390934 | pubmed:abstractText | Reperfusion injury (RI), a potential life-threatening disorder, represents an acute inflammatory response after periods of ischemia resulting from myocardial infarction, stroke, surgery, or trauma. The recent identification of a monoclonal natural IgM that initiates RI led to the identification of nonmuscle myosin heavy chain type II A and C as the self-targets in two different tissues. These results identify a novel pathway in which the innate response to a highly conserved self-antigen expressed as a result of hypoxic stress results in tissue destruction. | lld:pubmed |
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| pubmed-article:16390934 | pubmed:language | eng | lld:pubmed |
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| pubmed-article:16390934 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:16390934 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:16390934 | pubmed:month | Jan | lld:pubmed |
| pubmed-article:16390934 | pubmed:issn | 0022-1007 | lld:pubmed |
| pubmed-article:16390934 | pubmed:author | pubmed-author:MooreFrancis... | lld:pubmed |
| pubmed-article:16390934 | pubmed:author | pubmed-author:WeisslederRal... | lld:pubmed |
| pubmed-article:16390934 | pubmed:author | pubmed-author:ShimaokaMotom... | lld:pubmed |
| pubmed-article:16390934 | pubmed:author | pubmed-author:CarrollMichae... | lld:pubmed |
| pubmed-article:16390934 | pubmed:author | pubmed-author:VernaNicolaN | lld:pubmed |
| pubmed-article:16390934 | pubmed:author | pubmed-author:ZhangMingM | lld:pubmed |
| pubmed-article:16390934 | pubmed:author | pubmed-author:MahmoodUmarU | lld:pubmed |
| pubmed-article:16390934 | pubmed:author | pubmed-author:KesslerBenedi... | lld:pubmed |
| pubmed-article:16390934 | pubmed:author | pubmed-author:Vorup-JensenT... | lld:pubmed |
| pubmed-article:16390934 | pubmed:author | pubmed-author:ChiuIsaacI | lld:pubmed |
| pubmed-article:16390934 | pubmed:author | pubmed-author:AlicotElisabe... | lld:pubmed |
| pubmed-article:16390934 | pubmed:author | pubmed-author:ChanRodneyR | lld:pubmed |
| pubmed-article:16390934 | pubmed:author | pubmed-author:FriendDanielD | lld:pubmed |
| pubmed-article:16390934 | pubmed:author | pubmed-author:LiJinanJ | lld:pubmed |
| pubmed-article:16390934 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:16390934 | pubmed:day | 23 | lld:pubmed |
| pubmed-article:16390934 | pubmed:volume | 203 | lld:pubmed |
| pubmed-article:16390934 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:16390934 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:16390934 | pubmed:pagination | 141-52 | lld:pubmed |
| pubmed-article:16390934 | pubmed:dateRevised | 2009-11-18 | lld:pubmed |
| pubmed-article:16390934 | pubmed:meshHeading | pubmed-meshheading:16390934... | lld:pubmed |
| pubmed-article:16390934 | pubmed:meshHeading | pubmed-meshheading:16390934... | lld:pubmed |
