| pubmed-article:16380549 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:16380549 | lifeskim:mentions | umls-concept:C0003483 | lld:lifeskim |
| pubmed-article:16380549 | lifeskim:mentions | umls-concept:C0003009 | lld:lifeskim |
| pubmed-article:16380549 | lifeskim:mentions | umls-concept:C1383860 | lld:lifeskim |
| pubmed-article:16380549 | lifeskim:mentions | umls-concept:C0574032 | lld:lifeskim |
| pubmed-article:16380549 | lifeskim:mentions | umls-concept:C2245038 | lld:lifeskim |
| pubmed-article:16380549 | lifeskim:mentions | umls-concept:C0185014 | lld:lifeskim |
| pubmed-article:16380549 | lifeskim:mentions | umls-concept:C0205263 | lld:lifeskim |
| pubmed-article:16380549 | lifeskim:mentions | umls-concept:C1533157 | lld:lifeskim |
| pubmed-article:16380549 | pubmed:issue | 1 | lld:pubmed |
| pubmed-article:16380549 | pubmed:dateCreated | 2006-1-4 | lld:pubmed |
| pubmed-article:16380549 | pubmed:abstractText | A number of distinct stress signaling pathways in myocardium cause cardiac hypertrophy and heart failure. Class II histone deacetylases (HDACs) antagonize several stress-induced pathways and hypertrophy. However, cardiac hypertrophy induced by transgenic overexpression of the homeodomain only protein, HOP, can be prevented by the nonspecific HDAC inhibitors trichostatin A and valproic acid, suggesting that alternate targets that oppose class II HDAC function might exist in myocardium. We tested the effects of several HDAC inhibitors, including a class I HDAC-selective inhibitor, SK-7041, on cardiac hypertrophy induced by angiotensin II (Ang II) treatment or aortic banding (AB). | lld:pubmed |
| pubmed-article:16380549 | pubmed:language | eng | lld:pubmed |
| pubmed-article:16380549 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:16380549 | pubmed:citationSubset | AIM | lld:pubmed |
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| pubmed-article:16380549 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:16380549 | pubmed:month | Jan | lld:pubmed |
| pubmed-article:16380549 | pubmed:issn | 1524-4539 | lld:pubmed |
| pubmed-article:16380549 | pubmed:author | pubmed-author:TseLL | lld:pubmed |
| pubmed-article:16380549 | pubmed:author | pubmed-author:KimJong-KeunJ... | lld:pubmed |
| pubmed-article:16380549 | pubmed:author | pubmed-author:EpsteinJonath... | lld:pubmed |
| pubmed-article:16380549 | pubmed:author | pubmed-author:KimKyung... | lld:pubmed |
| pubmed-article:16380549 | pubmed:author | pubmed-author:KimNacksungN | lld:pubmed |
| pubmed-article:16380549 | pubmed:author | pubmed-author:BangYung-JueY... | lld:pubmed |
| pubmed-article:16380549 | pubmed:author | pubmed-author:ZhenYunY | lld:pubmed |
| pubmed-article:16380549 | pubmed:author | pubmed-author:NamKwang IlKI | lld:pubmed |
| pubmed-article:16380549 | pubmed:author | pubmed-author:KeeHae JinHJ | lld:pubmed |
| pubmed-article:16380549 | pubmed:author | pubmed-author:AhnYoungkeunY | lld:pubmed |
| pubmed-article:16380549 | pubmed:author | pubmed-author:JeongMyung... | lld:pubmed |
| pubmed-article:16380549 | pubmed:author | pubmed-author:ParkJong... | lld:pubmed |
| pubmed-article:16380549 | pubmed:author | pubmed-author:SohnIl SukIS | lld:pubmed |
| pubmed-article:16380549 | pubmed:author | pubmed-author:QianYong RiYR | lld:pubmed |
| pubmed-article:16380549 | pubmed:issnType | Electronic | lld:pubmed |
| pubmed-article:16380549 | pubmed:day | 3 | lld:pubmed |
| pubmed-article:16380549 | pubmed:volume | 113 | lld:pubmed |
| pubmed-article:16380549 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:16380549 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:16380549 | pubmed:pagination | 51-9 | lld:pubmed |
| pubmed-article:16380549 | pubmed:dateRevised | 2009-11-19 | lld:pubmed |
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| pubmed-article:16380549 | pubmed:year | 2006 | lld:pubmed |
| pubmed-article:16380549 | pubmed:articleTitle | Inhibition of histone deacetylation blocks cardiac hypertrophy induced by angiotensin II infusion and aortic banding. | lld:pubmed |
| pubmed-article:16380549 | pubmed:affiliation | Department of Pharmacology, Research Institute of Medical Sciences and Medical Research Center for Gene Regulation, Chonnam National University Medical School, Gwangju, South Korea. | lld:pubmed |
| pubmed-article:16380549 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:16380549 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
| pubmed-article:16380549 | pubmed:publicationType | Research Support, N.I.H., Extramural | lld:pubmed |
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