| pubmed-article:16360962 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:16360962 | lifeskim:mentions | umls-concept:C0035820 | lld:lifeskim |
| pubmed-article:16360962 | lifeskim:mentions | umls-concept:C0018801 | lld:lifeskim |
| pubmed-article:16360962 | lifeskim:mentions | umls-concept:C0002651 | lld:lifeskim |
| pubmed-article:16360962 | lifeskim:mentions | umls-concept:C1274040 | lld:lifeskim |
| pubmed-article:16360962 | lifeskim:mentions | umls-concept:C0475224 | lld:lifeskim |
| pubmed-article:16360962 | lifeskim:mentions | umls-concept:C1882417 | lld:lifeskim |
| pubmed-article:16360962 | lifeskim:mentions | umls-concept:C0205214 | lld:lifeskim |
| pubmed-article:16360962 | pubmed:issue | 9 | lld:pubmed |
| pubmed-article:16360962 | pubmed:dateCreated | 2005-12-19 | lld:pubmed |
| pubmed-article:16360962 | pubmed:abstractText | A common variant of the adenosine monophosphate deaminase (AMPD)-1 gene (C34T) results in enzymatic inactivity and may increase adenosine in cardiac muscle and confer cardioprotection through ischemic preconditioning. | lld:pubmed |
| pubmed-article:16360962 | pubmed:language | eng | lld:pubmed |
| pubmed-article:16360962 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:16360962 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:16360962 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:16360962 | pubmed:month | Dec | lld:pubmed |
| pubmed-article:16360962 | pubmed:issn | 1071-9164 | lld:pubmed |
| pubmed-article:16360962 | pubmed:author | pubmed-author:AndersonJeffr... | lld:pubmed |
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| pubmed-article:16360962 | pubmed:author | pubmed-author:CarlquistJohn... | lld:pubmed |
| pubmed-article:16360962 | pubmed:author | pubmed-author:KolekMatthew... | lld:pubmed |
| pubmed-article:16360962 | pubmed:author | pubmed-author:WhitingBryant... | lld:pubmed |
| pubmed-article:16360962 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:16360962 | pubmed:volume | 11 | lld:pubmed |
| pubmed-article:16360962 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:16360962 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:16360962 | pubmed:pagination | 677-83 | lld:pubmed |
| pubmed-article:16360962 | pubmed:dateRevised | 2007-11-15 | lld:pubmed |
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| pubmed-article:16360962 | pubmed:year | 2005 | lld:pubmed |
| pubmed-article:16360962 | pubmed:articleTitle | The role of a common adenosine monophosphate deaminase (AMPD)-1 polymorphism in outcomes of ischemic and nonischemic heart failure. | lld:pubmed |
| pubmed-article:16360962 | pubmed:affiliation | Cardiovascular Department, LDS Hospital, Salt Lake City, UT 84143, USA. | lld:pubmed |
| pubmed-article:16360962 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:16360962 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
| pubmed-article:16360962 | pubmed:publicationType | Multicenter Study | lld:pubmed |
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| http://linkedlifedata.com/r... | pubmed:referesTo | pubmed-article:16360962 | lld:pubmed |
