| pubmed-article:16301341 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:16301341 | lifeskim:mentions | umls-concept:C0035820 | lld:lifeskim |
| pubmed-article:16301341 | lifeskim:mentions | umls-concept:C0014653 | lld:lifeskim |
| pubmed-article:16301341 | lifeskim:mentions | umls-concept:C0027051 | lld:lifeskim |
| pubmed-article:16301341 | lifeskim:mentions | umls-concept:C0018787 | lld:lifeskim |
| pubmed-article:16301341 | lifeskim:mentions | umls-concept:C0669368 | lld:lifeskim |
| pubmed-article:16301341 | lifeskim:mentions | umls-concept:C0026565 | lld:lifeskim |
| pubmed-article:16301341 | lifeskim:mentions | umls-concept:C0442805 | lld:lifeskim |
| pubmed-article:16301341 | lifeskim:mentions | umls-concept:C0011155 | lld:lifeskim |
| pubmed-article:16301341 | pubmed:issue | 22 | lld:pubmed |
| pubmed-article:16301341 | pubmed:dateCreated | 2005-11-30 | lld:pubmed |
| pubmed-article:16301341 | pubmed:abstractText | Neuronal nitric oxide synthase (NOS1) plays key cardiac physiological roles, regulating excitation-contraction coupling and exerting an antioxidant effect that maintains tissue NO-redox equilibrium. After myocardial infarction (MI), NOS1 translocates from the sarcoplasmic reticulum to the cell membrane, where it inhibits beta-adrenergic contractility, an effect previously predicted to have adverse consequences. Counter to this idea, we tested the hypothesis that NOS1 has a protective effect after MI. | lld:pubmed |
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| pubmed-article:16301341 | pubmed:language | eng | lld:pubmed |
| pubmed-article:16301341 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:16301341 | pubmed:citationSubset | AIM | lld:pubmed |
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| pubmed-article:16301341 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:16301341 | pubmed:month | Nov | lld:pubmed |
| pubmed-article:16301341 | pubmed:issn | 1524-4539 | lld:pubmed |
| pubmed-article:16301341 | pubmed:author | pubmed-author:LiDechunD | lld:pubmed |
| pubmed-article:16301341 | pubmed:author | pubmed-author:HareJoshua... | lld:pubmed |
| pubmed-article:16301341 | pubmed:author | pubmed-author:BarouchLili... | lld:pubmed |
| pubmed-article:16301341 | pubmed:author | pubmed-author:RajuShubha... | lld:pubmed |
| pubmed-article:16301341 | pubmed:author | pubmed-author:SaraivaRobert... | lld:pubmed |
| pubmed-article:16301341 | pubmed:author | pubmed-author:MinhasKhalid... | lld:pubmed |
| pubmed-article:16301341 | pubmed:author | pubmed-author:SchuleriKarl... | lld:pubmed |
| pubmed-article:16301341 | pubmed:author | pubmed-author:PitzEleanorE | lld:pubmed |
| pubmed-article:16301341 | pubmed:author | pubmed-author:VandegaerKoen... | lld:pubmed |
| pubmed-article:16301341 | pubmed:issnType | Electronic | lld:pubmed |
| pubmed-article:16301341 | pubmed:day | 29 | lld:pubmed |
| pubmed-article:16301341 | pubmed:volume | 112 | lld:pubmed |
| pubmed-article:16301341 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:16301341 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:16301341 | pubmed:pagination | 3415-22 | lld:pubmed |
| pubmed-article:16301341 | pubmed:dateRevised | 2007-11-14 | lld:pubmed |
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| pubmed-article:16301341 | pubmed:meshHeading | pubmed-meshheading:16301341... | lld:pubmed |
| pubmed-article:16301341 | pubmed:year | 2005 | lld:pubmed |
| pubmed-article:16301341 | pubmed:articleTitle | Deficiency of neuronal nitric oxide synthase increases mortality and cardiac remodeling after myocardial infarction: role of nitroso-redox equilibrium. | lld:pubmed |
| pubmed-article:16301341 | pubmed:affiliation | Cardiology Division, Department of Medicine, Johns Hopkins Medical Institutions, Baltimore, MD 21205, USA. | lld:pubmed |
| pubmed-article:16301341 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:16301341 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
| pubmed-article:16301341 | pubmed:publicationType | Research Support, N.I.H., Extramural | lld:pubmed |
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