| pubmed-article:16216978 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:16216978 | lifeskim:mentions | umls-concept:C0026809 | lld:lifeskim |
| pubmed-article:16216978 | lifeskim:mentions | umls-concept:C0018787 | lld:lifeskim |
| pubmed-article:16216978 | lifeskim:mentions | umls-concept:C1522564 | lld:lifeskim |
| pubmed-article:16216978 | lifeskim:mentions | umls-concept:C0027481 | lld:lifeskim |
| pubmed-article:16216978 | lifeskim:mentions | umls-concept:C1707919 | lld:lifeskim |
| pubmed-article:16216978 | lifeskim:mentions | umls-concept:C1621574 | lld:lifeskim |
| pubmed-article:16216978 | lifeskim:mentions | umls-concept:C0597484 | lld:lifeskim |
| pubmed-article:16216978 | lifeskim:mentions | umls-concept:C1880177 | lld:lifeskim |
| pubmed-article:16216978 | pubmed:issue | 15 | lld:pubmed |
| pubmed-article:16216978 | pubmed:dateCreated | 2005-10-11 | lld:pubmed |
| pubmed-article:16216978 | pubmed:abstractText | Atrial natriuretic peptide (ANP), through its guanylyl cyclase-A (GC-A) receptor, not only is critically involved in the endocrine regulation of arterial blood pressure but also locally moderates cardiomyocyte growth. The mechanisms underlying the antihypertrophic effects of ANP remain largely uncharacterized. We examined the contribution of the Na+/H+ exchanger NHE-1 to cardiac remodeling in GC-A-deficient (GC-A(-/-)) mice. | lld:pubmed |
| pubmed-article:16216978 | pubmed:language | eng | lld:pubmed |
| pubmed-article:16216978 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:16216978 | pubmed:citationSubset | AIM | lld:pubmed |
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| pubmed-article:16216978 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:16216978 | pubmed:month | Oct | lld:pubmed |
| pubmed-article:16216978 | pubmed:issn | 1524-4539 | lld:pubmed |
| pubmed-article:16216978 | pubmed:author | pubmed-author:BabaHideo AHA | lld:pubmed |
| pubmed-article:16216978 | pubmed:author | pubmed-author:van... | lld:pubmed |
| pubmed-article:16216978 | pubmed:author | pubmed-author:KuhnMichaelaM | lld:pubmed |
| pubmed-article:16216978 | pubmed:author | pubmed-author:SchlatterEber... | lld:pubmed |
| pubmed-article:16216978 | pubmed:author | pubmed-author:FabritzLariss... | lld:pubmed |
| pubmed-article:16216978 | pubmed:author | pubmed-author:VelicAnaA | lld:pubmed |
| pubmed-article:16216978 | pubmed:author | pubmed-author:VossMelanieM | lld:pubmed |
| pubmed-article:16216978 | pubmed:author | pubmed-author:KilicAnaA | lld:pubmed |
| pubmed-article:16216978 | pubmed:author | pubmed-author:MitkoDanutaD | lld:pubmed |
| pubmed-article:16216978 | pubmed:author | pubmed-author:De... | lld:pubmed |
| pubmed-article:16216978 | pubmed:author | pubmed-author:ZwienerMelani... | lld:pubmed |
| pubmed-article:16216978 | pubmed:issnType | Electronic | lld:pubmed |
| pubmed-article:16216978 | pubmed:day | 11 | lld:pubmed |
| pubmed-article:16216978 | pubmed:volume | 112 | lld:pubmed |
| pubmed-article:16216978 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:16216978 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:16216978 | pubmed:pagination | 2307-17 | lld:pubmed |
| pubmed-article:16216978 | pubmed:dateRevised | 2007-11-15 | lld:pubmed |
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| pubmed-article:16216978 | pubmed:meshHeading | pubmed-meshheading:16216978... | lld:pubmed |
| pubmed-article:16216978 | pubmed:year | 2005 | lld:pubmed |
| pubmed-article:16216978 | pubmed:articleTitle | Enhanced activity of the myocardial Na+/H+ exchanger NHE-1 contributes to cardiac remodeling in atrial natriuretic peptide receptor-deficient mice. | lld:pubmed |
| pubmed-article:16216978 | pubmed:affiliation | Institute of Physiology, University of Würzburg, Würzburg, Germany. | lld:pubmed |
| pubmed-article:16216978 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:16216978 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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