pubmed-article:16204373 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:16204373 | lifeskim:mentions | umls-concept:C0086418 | lld:lifeskim |
pubmed-article:16204373 | lifeskim:mentions | umls-concept:C0022131 | lld:lifeskim |
pubmed-article:16204373 | lifeskim:mentions | umls-concept:C0031621 | lld:lifeskim |
pubmed-article:16204373 | lifeskim:mentions | umls-concept:C0063684 | lld:lifeskim |
pubmed-article:16204373 | lifeskim:mentions | umls-concept:C0600688 | lld:lifeskim |
pubmed-article:16204373 | lifeskim:mentions | umls-concept:C1335238 | lld:lifeskim |
pubmed-article:16204373 | lifeskim:mentions | umls-concept:C0289313 | lld:lifeskim |
pubmed-article:16204373 | lifeskim:mentions | umls-concept:C1415831 | lld:lifeskim |
pubmed-article:16204373 | lifeskim:mentions | umls-concept:C1704259 | lld:lifeskim |
pubmed-article:16204373 | lifeskim:mentions | umls-concept:C1705987 | lld:lifeskim |
pubmed-article:16204373 | lifeskim:mentions | umls-concept:C1879547 | lld:lifeskim |
pubmed-article:16204373 | pubmed:issue | 12 | lld:pubmed |
pubmed-article:16204373 | pubmed:dateCreated | 2005-12-6 | lld:pubmed |
pubmed-article:16204373 | pubmed:abstractText | Type 2 diabetes mellitus (T2DM) is characterized by a deficit in beta-cell mass, increased beta-cell apoptosis, and islet amyloid derived from islet amyloid polypeptide (IAPP). Human IAPP (h-IAPP) applied to beta-cells forms toxic oligomers that induce apoptosis. Thiazolidinediones, ligands of peroxisome proliferator-activated receptor-gamma (PPAR-gamma), can delay the onset of T2DM. | lld:pubmed |
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pubmed-article:16204373 | pubmed:language | eng | lld:pubmed |
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pubmed-article:16204373 | pubmed:citationSubset | AIM | lld:pubmed |
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pubmed-article:16204373 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:16204373 | pubmed:month | Dec | lld:pubmed |
pubmed-article:16204373 | pubmed:issn | 0021-972X | lld:pubmed |
pubmed-article:16204373 | pubmed:author | pubmed-author:HaatajaLeenaL | lld:pubmed |
pubmed-article:16204373 | pubmed:author | pubmed-author:ButlerPeter... | lld:pubmed |
pubmed-article:16204373 | pubmed:author | pubmed-author:HsuehWilla... | lld:pubmed |
pubmed-article:16204373 | pubmed:author | pubmed-author:LinChia-YuCY | lld:pubmed |
pubmed-article:16204373 | pubmed:author | pubmed-author:GurloTatyanaT | lld:pubmed |
pubmed-article:16204373 | pubmed:issnType | Print | lld:pubmed |
pubmed-article:16204373 | pubmed:volume | 90 | lld:pubmed |
pubmed-article:16204373 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:16204373 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:16204373 | pubmed:pagination | 6678-86 | lld:pubmed |
pubmed-article:16204373 | pubmed:dateRevised | 2011-11-17 | lld:pubmed |
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pubmed-article:16204373 | pubmed:year | 2005 | lld:pubmed |
pubmed-article:16204373 | pubmed:articleTitle | Activation of peroxisome proliferator-activated receptor-gamma by rosiglitazone protects human islet cells against human islet amyloid polypeptide toxicity by a phosphatidylinositol 3'-kinase-dependent pathway. | lld:pubmed |
pubmed-article:16204373 | pubmed:affiliation | Larry Hillblom Islet Research Center, Division of Endocrinology, David Geffen School of Medicine at University of California Los Angeles, 900A Weyburn Place North, Los Angeles, California 90095-7073, USA. | lld:pubmed |
pubmed-article:16204373 | pubmed:publicationType | Journal Article | lld:pubmed |
pubmed-article:16204373 | pubmed:publicationType | In Vitro | lld:pubmed |
pubmed-article:16204373 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
pubmed-article:16204373 | pubmed:publicationType | Research Support, N.I.H., Extramural | lld:pubmed |
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