pubmed-article:16204299 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:16204299 | lifeskim:mentions | umls-concept:C0026336 | lld:lifeskim |
pubmed-article:16204299 | lifeskim:mentions | umls-concept:C1882714 | lld:lifeskim |
pubmed-article:16204299 | lifeskim:mentions | umls-concept:C0205409 | lld:lifeskim |
pubmed-article:16204299 | lifeskim:mentions | umls-concept:C0599732 | lld:lifeskim |
pubmed-article:16204299 | lifeskim:mentions | umls-concept:C0392756 | lld:lifeskim |
pubmed-article:16204299 | lifeskim:mentions | umls-concept:C2266987 | lld:lifeskim |
pubmed-article:16204299 | lifeskim:mentions | umls-concept:C1549542 | lld:lifeskim |
pubmed-article:16204299 | pubmed:issue | 12 | lld:pubmed |
pubmed-article:16204299 | pubmed:dateCreated | 2005-11-18 | lld:pubmed |
pubmed-article:16204299 | pubmed:abstractText | In vitro, cold-induced injury is an important contributor to renal tubular cell damage. It is mediated by iron-dependent formation of reactive oxygen species and can be prevented by iron chelation. We studied whether iron chelators can prevent cold-induced damage in the isolated perfused rat kidney (IPK) model both after cold perfusion (CP) and after cold storage (CS). We hypothesized that in the CP model iron-dependent cold-induced injury is more pronounced, since oxygen is constantly provided. | lld:pubmed |
pubmed-article:16204299 | pubmed:language | eng | lld:pubmed |
pubmed-article:16204299 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:16204299 | pubmed:citationSubset | IM | lld:pubmed |
pubmed-article:16204299 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:16204299 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:16204299 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:16204299 | pubmed:month | Dec | lld:pubmed |
pubmed-article:16204299 | pubmed:issn | 0931-0509 | lld:pubmed |
pubmed-article:16204299 | pubmed:author | pubmed-author:KramersCornel... | lld:pubmed |
pubmed-article:16204299 | pubmed:author | pubmed-author:RusselFrans... | lld:pubmed |
pubmed-article:16204299 | pubmed:author | pubmed-author:SteenbergenEr... | lld:pubmed |
pubmed-article:16204299 | pubmed:author | pubmed-author:WetzelsJack... | lld:pubmed |
pubmed-article:16204299 | pubmed:author | pubmed-author:Bartels-Strin... | lld:pubmed |
pubmed-article:16204299 | pubmed:author | pubmed-author:WouterseAlfon... | lld:pubmed |
pubmed-article:16204299 | pubmed:issnType | Print | lld:pubmed |
pubmed-article:16204299 | pubmed:volume | 20 | lld:pubmed |
pubmed-article:16204299 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:16204299 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:16204299 | pubmed:pagination | 2646-53 | lld:pubmed |
pubmed-article:16204299 | pubmed:dateRevised | 2006-11-15 | lld:pubmed |
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pubmed-article:16204299 | pubmed:year | 2005 | lld:pubmed |
pubmed-article:16204299 | pubmed:articleTitle | Iron chelators do not reduce cold-induced cell injury in the isolated perfused rat kidney model. | lld:pubmed |
pubmed-article:16204299 | pubmed:affiliation | Department of Pharmacology and Toxicology, Radboud University Nijmegen Medical Centre, P.O. Box 9101, 6500 HB Nijmegen, The Netherlands. | lld:pubmed |
pubmed-article:16204299 | pubmed:publicationType | Journal Article | lld:pubmed |
pubmed-article:16204299 | pubmed:publicationType | Comparative Study | lld:pubmed |
pubmed-article:16204299 | pubmed:publicationType | In Vitro | lld:pubmed |