pubmed-article:16189096 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:16189096 | lifeskim:mentions | umls-concept:C0038410 | lld:lifeskim |
pubmed-article:16189096 | lifeskim:mentions | umls-concept:C0282563 | lld:lifeskim |
pubmed-article:16189096 | lifeskim:mentions | umls-concept:C0086345 | lld:lifeskim |
pubmed-article:16189096 | lifeskim:mentions | umls-concept:C0033105 | lld:lifeskim |
pubmed-article:16189096 | lifeskim:mentions | umls-concept:C0370215 | lld:lifeskim |
pubmed-article:16189096 | lifeskim:mentions | umls-concept:C0683598 | lld:lifeskim |
pubmed-article:16189096 | lifeskim:mentions | umls-concept:C0016132 | lld:lifeskim |
pubmed-article:16189096 | lifeskim:mentions | umls-concept:C1516695 | lld:lifeskim |
pubmed-article:16189096 | pubmed:issue | 10 | lld:pubmed |
pubmed-article:16189096 | pubmed:dateCreated | 2005-9-28 | lld:pubmed |
pubmed-article:16189096 | pubmed:abstractText | The prevalence and mechanisms of macrolide resistance among 1,007 clinical pneumococcal isolates collected in Finland were investigated. Of these, 217 (21.5%) were resistant to erythromycin and 11% to clindamycin. Among the erythromycin-resistant isolates, mef(E) was present in 95 isolates (44%), mef(A) was present in 12 isolates (6%), and erm(B) was present in 90 isolates (41%). A double mechanism, mef(E) and erm(B), was detected in five isolates (2%). Ribosomal mutation was detected in 14 (6%) macrolide-resistant isolates in which no other determinant was found. Based on the telithromycin MICs, two groups of isolates were formed: 83.3% of the isolates belonged to a major group for which the telithromycin MIC range was < or =0.008 to 0.063 microg/ml, and 16.7% belonged to a minor group for which the telithromycin MIC range was 0.125 to 8 microg/ml. All except three isolates in the minor population carried a macrolide resistance gene. | lld:pubmed |
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pubmed-article:16189096 | pubmed:language | eng | lld:pubmed |
pubmed-article:16189096 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:16189096 | pubmed:citationSubset | IM | lld:pubmed |
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pubmed-article:16189096 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:16189096 | pubmed:month | Oct | lld:pubmed |
pubmed-article:16189096 | pubmed:issn | 0066-4804 | lld:pubmed |
pubmed-article:16189096 | pubmed:author | pubmed-author:HuovinenPP | lld:pubmed |
pubmed-article:16189096 | pubmed:author | pubmed-author:RantalaMM | lld:pubmed |
pubmed-article:16189096 | pubmed:author | pubmed-author:JalavaJJ | lld:pubmed |
pubmed-article:16189096 | pubmed:author | pubmed-author:HuikkoSS | lld:pubmed |
pubmed-article:16189096 | pubmed:issnType | Print | lld:pubmed |
pubmed-article:16189096 | pubmed:volume | 49 | lld:pubmed |
pubmed-article:16189096 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:16189096 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:16189096 | pubmed:pagination | 4180-4 | lld:pubmed |
pubmed-article:16189096 | pubmed:dateRevised | 2009-11-18 | lld:pubmed |
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pubmed-article:16189096 | pubmed:year | 2005 | lld:pubmed |
pubmed-article:16189096 | pubmed:articleTitle | Prevalence and molecular genetics of macrolide resistance among Streptococcus pneumoniae isolates collected in Finland in 2002. | lld:pubmed |
pubmed-article:16189096 | pubmed:affiliation | Department of Bacteriology and Inflammation, National Public Health Institute, Turku, Finland. merja.rantala@ktl.fi | lld:pubmed |
pubmed-article:16189096 | pubmed:publicationType | Journal Article | lld:pubmed |
pubmed-article:16189096 | pubmed:publicationType | Comparative Study | lld:pubmed |
pubmed-article:16189096 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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