pubmed-article:16168521 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:16168521 | lifeskim:mentions | umls-concept:C0684336 | lld:lifeskim |
pubmed-article:16168521 | lifeskim:mentions | umls-concept:C0086418 | lld:lifeskim |
pubmed-article:16168521 | lifeskim:mentions | umls-concept:C2239176 | lld:lifeskim |
pubmed-article:16168521 | lifeskim:mentions | umls-concept:C0022688 | lld:lifeskim |
pubmed-article:16168521 | lifeskim:mentions | umls-concept:C0011306 | lld:lifeskim |
pubmed-article:16168521 | lifeskim:mentions | umls-concept:C0007613 | lld:lifeskim |
pubmed-article:16168521 | lifeskim:mentions | umls-concept:C1749467 | lld:lifeskim |
pubmed-article:16168521 | lifeskim:mentions | umls-concept:C0376315 | lld:lifeskim |
pubmed-article:16168521 | lifeskim:mentions | umls-concept:C0205179 | lld:lifeskim |
pubmed-article:16168521 | lifeskim:mentions | umls-concept:C0389061 | lld:lifeskim |
pubmed-article:16168521 | pubmed:issue | 6 | lld:pubmed |
pubmed-article:16168521 | pubmed:dateCreated | 2005-11-21 | lld:pubmed |
pubmed-article:16168521 | pubmed:abstractText | MHC class I-related chain A (MICA), a human ligand of natural killer (NK) cell stimulatory receptor NKG2D, is expressed in human hepatocellular carcinomas (HCC). Earlier research demonstrated that the soluble form of MICA (sMICA) is released from some types of tumors, but its presence and role in HCC was not determined. | lld:pubmed |
pubmed-article:16168521 | pubmed:language | eng | lld:pubmed |
pubmed-article:16168521 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:16168521 | pubmed:citationSubset | IM | lld:pubmed |
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pubmed-article:16168521 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:16168521 | pubmed:month | Dec | lld:pubmed |
pubmed-article:16168521 | pubmed:issn | 0168-8278 | lld:pubmed |
pubmed-article:16168521 | pubmed:author | pubmed-author:HayashiNorioN | lld:pubmed |
pubmed-article:16168521 | pubmed:author | pubmed-author:TatsumiTomohi... | lld:pubmed |
pubmed-article:16168521 | pubmed:author | pubmed-author:YamaguchiShin... | lld:pubmed |
pubmed-article:16168521 | pubmed:author | pubmed-author:TakeharaTetsu... | lld:pubmed |
pubmed-article:16168521 | pubmed:author | pubmed-author:JinushiMasahi... | lld:pubmed |
pubmed-article:16168521 | pubmed:author | pubmed-author:HiramatsuNaok... | lld:pubmed |
pubmed-article:16168521 | pubmed:author | pubmed-author:SakamoriRyota... | lld:pubmed |
pubmed-article:16168521 | pubmed:issnType | Print | lld:pubmed |
pubmed-article:16168521 | pubmed:volume | 43 | lld:pubmed |
pubmed-article:16168521 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:16168521 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:16168521 | pubmed:pagination | 1013-20 | lld:pubmed |
pubmed-article:16168521 | pubmed:dateRevised | 2008-11-21 | lld:pubmed |
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pubmed-article:16168521 | pubmed:year | 2005 | lld:pubmed |
pubmed-article:16168521 | pubmed:articleTitle | Impairment of natural killer cell and dendritic cell functions by the soluble form of MHC class I-related chain A in advanced human hepatocellular carcinomas. | lld:pubmed |
pubmed-article:16168521 | pubmed:affiliation | Department of Molecular Therapeutics, Graduate School of Medicine, Osaka University, 2-2 Yamada-oka, Suita, Osaka 565-0871, Japan. | lld:pubmed |
pubmed-article:16168521 | pubmed:publicationType | Journal Article | lld:pubmed |
pubmed-article:16168521 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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