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| pubmed-article:16157899 | lifeskim:mentions | umls-concept:C0026339 | lld:lifeskim |
| pubmed-article:16157899 | lifeskim:mentions | umls-concept:C0025914 | lld:lifeskim |
| pubmed-article:16157899 | lifeskim:mentions | umls-concept:C0026809 | lld:lifeskim |
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| pubmed-article:16157899 | lifeskim:mentions | umls-concept:C2698698 | lld:lifeskim |
| pubmed-article:16157899 | lifeskim:mentions | umls-concept:C0270911 | lld:lifeskim |
| pubmed-article:16157899 | lifeskim:mentions | umls-concept:C0027756 | lld:lifeskim |
| pubmed-article:16157899 | lifeskim:mentions | umls-concept:C0033414 | lld:lifeskim |
| pubmed-article:16157899 | lifeskim:mentions | umls-concept:C1413535 | lld:lifeskim |
| pubmed-article:16157899 | lifeskim:mentions | umls-concept:C0445254 | lld:lifeskim |
| pubmed-article:16157899 | pubmed:issue | 5 | lld:pubmed |
| pubmed-article:16157899 | pubmed:dateCreated | 2005-9-13 | lld:pubmed |
| pubmed-article:16157899 | pubmed:abstractText | Xenografts from patients with Charcot-Marie-Tooth type 1A (CMT1A) have shown delayed myelination and impaired regeneration of nude mice axons passing through the grafted segments. Neurotrophin-3 (NT-3), an important component of the Schwann cell (SC) autocrine survival loop, could correct these deficiencies. | lld:pubmed |
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| pubmed-article:16157899 | pubmed:language | eng | lld:pubmed |
| pubmed-article:16157899 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:16157899 | pubmed:citationSubset | AIM | lld:pubmed |
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| pubmed-article:16157899 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:16157899 | pubmed:month | Sep | lld:pubmed |
| pubmed-article:16157899 | pubmed:issn | 1526-632X | lld:pubmed |
| pubmed-article:16157899 | pubmed:author | pubmed-author:CedarbaumJ... | lld:pubmed |
| pubmed-article:16157899 | pubmed:author | pubmed-author:KingW MWM | lld:pubmed |
| pubmed-article:16157899 | pubmed:author | pubmed-author:MendellJ RJR | lld:pubmed |
| pubmed-article:16157899 | pubmed:author | pubmed-author:SahenkZZ | lld:pubmed |
| pubmed-article:16157899 | pubmed:author | pubmed-author:FreimerM LML | lld:pubmed |
| pubmed-article:16157899 | pubmed:author | pubmed-author:NagarajaH NHN | lld:pubmed |
| pubmed-article:16157899 | pubmed:author | pubmed-author:McCrackenB... | lld:pubmed |
| pubmed-article:16157899 | pubmed:issnType | Electronic | lld:pubmed |
| pubmed-article:16157899 | pubmed:day | 13 | lld:pubmed |
| pubmed-article:16157899 | pubmed:volume | 65 | lld:pubmed |
| pubmed-article:16157899 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:16157899 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:16157899 | pubmed:pagination | 681-9 | lld:pubmed |
| pubmed-article:16157899 | pubmed:dateRevised | 2007-11-14 | lld:pubmed |
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| pubmed-article:16157899 | pubmed:year | 2005 | lld:pubmed |
| pubmed-article:16157899 | pubmed:articleTitle | NT-3 promotes nerve regeneration and sensory improvement in CMT1A mouse models and in patients. | lld:pubmed |
| pubmed-article:16157899 | pubmed:affiliation | Department of Neurology, Ohio State University, Columbus, OH, USA. Sahenk.1@osu.edu | lld:pubmed |
| pubmed-article:16157899 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:16157899 | pubmed:publicationType | Clinical Trial | lld:pubmed |
| pubmed-article:16157899 | pubmed:publicationType | Research Support, U.S. Gov't, P.H.S. | lld:pubmed |
| pubmed-article:16157899 | pubmed:publicationType | Randomized Controlled Trial | lld:pubmed |
| pubmed-article:16157899 | pubmed:publicationType | Research Support, N.I.H., Extramural | lld:pubmed |
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