16156900

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Subject	Predicate	Object	Context
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pubmed-article:16156900	lifeskim:mentions	umls-concept:C0042216	lld:lifeskim
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pubmed-article:16156900	lifeskim:mentions	umls-concept:C0017262	lld:lifeskim
pubmed-article:16156900	lifeskim:mentions	umls-concept:C1539341	lld:lifeskim
pubmed-article:16156900	lifeskim:mentions	umls-concept:C2587213	lld:lifeskim
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pubmed-article:16156900	lifeskim:mentions	umls-concept:C0140694	lld:lifeskim
pubmed-article:16156900	lifeskim:mentions	umls-concept:C0185117	lld:lifeskim
pubmed-article:16156900	pubmed:dateCreated	2005-10-6	lld:pubmed
pubmed-article:16156900	pubmed:abstractText	Hepatitis C virus (HCV) infection is of growing concern in public health with around 350 million chronically infected individuals worldwide. Although the IFN-alpha/rivabirin is the only approved therapy with 10-30% clinical efficacy, the protective molecular mechanism involved during the treatment is still unknown. To analyze the effect of HCV polyprotein expression on the antiviral response of the host, we developed a novel vaccinia virus (VV)-based delivery system (VT7-HCV7.9) where structural and nonstructural (except part of NS5B) proteins of HCV ORF from genotype 1b are efficiently expressed and produced, and timely regulated in mammalian cell lines.	lld:pubmed
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pubmed-article:16156900	pubmed:status	MEDLINE	lld:pubmed
pubmed-article:16156900	pubmed:issn	1743-422X	lld:pubmed
pubmed-article:16156900	pubmed:author	pubmed-author:EstebanMarian...	lld:pubmed
pubmed-article:16156900	pubmed:author	pubmed-author:GómezCarmen...	lld:pubmed
pubmed-article:16156900	pubmed:author	pubmed-author:VandermeerenA...	lld:pubmed

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