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| pubmed-article:16100013 | lifeskim:mentions | umls-concept:C0035820 | lld:lifeskim |
| pubmed-article:16100013 | lifeskim:mentions | umls-concept:C0036202 | lld:lifeskim |
| pubmed-article:16100013 | lifeskim:mentions | umls-concept:C0549493 | lld:lifeskim |
| pubmed-article:16100013 | lifeskim:mentions | umls-concept:C0699748 | lld:lifeskim |
| pubmed-article:16100013 | lifeskim:mentions | umls-concept:C1332824 | lld:lifeskim |
| pubmed-article:16100013 | lifeskim:mentions | umls-concept:C1424460 | lld:lifeskim |
| pubmed-article:16100013 | lifeskim:mentions | umls-concept:C0023688 | lld:lifeskim |
| pubmed-article:16100013 | pubmed:issue | 10 | lld:pubmed |
| pubmed-article:16100013 | pubmed:dateCreated | 2005-11-8 | lld:pubmed |
| pubmed-article:16100013 | pubmed:abstractText | Receptor expression dictates the spectrum of chemokine actions on immunocompetent cells. We have previously shown that the chemokine receptor CXCR3 is highly expressed by T-helper type 1 (Th1) cells infiltrating the lungs of patients with sarcoidosis. | lld:pubmed |
| pubmed-article:16100013 | pubmed:language | eng | lld:pubmed |
| pubmed-article:16100013 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:16100013 | pubmed:citationSubset | AIM | lld:pubmed |
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| pubmed-article:16100013 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:16100013 | pubmed:month | Nov | lld:pubmed |
| pubmed-article:16100013 | pubmed:issn | 1073-449X | lld:pubmed |
| pubmed-article:16100013 | pubmed:author | pubmed-author:ZambelloRenat... | lld:pubmed |
| pubmed-article:16100013 | pubmed:author | pubmed-author:AgostiniCarlo... | lld:pubmed |
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| pubmed-article:16100013 | pubmed:author | pubmed-author:CalabreseFior... | lld:pubmed |
| pubmed-article:16100013 | pubmed:author | pubmed-author:TrentinLivioL | lld:pubmed |
| pubmed-article:16100013 | pubmed:author | pubmed-author:FaccoMonicaM | lld:pubmed |
| pubmed-article:16100013 | pubmed:author | pubmed-author:CabrelleAnnaA | lld:pubmed |
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| pubmed-article:16100013 | pubmed:author | pubmed-author:CarraroSamuel... | lld:pubmed |
| pubmed-article:16100013 | pubmed:author | pubmed-author:BeghèBiancaB | lld:pubmed |
| pubmed-article:16100013 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:16100013 | pubmed:day | 15 | lld:pubmed |
| pubmed-article:16100013 | pubmed:volume | 172 | lld:pubmed |
| pubmed-article:16100013 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:16100013 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:16100013 | pubmed:pagination | 1290-8 | lld:pubmed |
| pubmed-article:16100013 | pubmed:dateRevised | 2006-11-15 | lld:pubmed |
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| pubmed-article:16100013 | pubmed:year | 2005 | lld:pubmed |
| pubmed-article:16100013 | pubmed:articleTitle | Role for CXCR6 and its ligand CXCL16 in the pathogenesis of T-cell alveolitis in sarcoidosis. | lld:pubmed |
| pubmed-article:16100013 | pubmed:affiliation | Department of Clinical Medicine, Clinical Immunology Branch, Division of Pneumology and Institute of Pathology, Padua University School of Medicine, Italy. carlo.agostini@unipd.it | lld:pubmed |
| pubmed-article:16100013 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:16100013 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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