pubmed-article:16034045 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:16034045 | lifeskim:mentions | umls-concept:C1333990 | lld:lifeskim |
pubmed-article:16034045 | lifeskim:mentions | umls-concept:C0017337 | lld:lifeskim |
pubmed-article:16034045 | lifeskim:mentions | umls-concept:C1155661 | lld:lifeskim |
pubmed-article:16034045 | lifeskim:mentions | umls-concept:C0021044 | lld:lifeskim |
pubmed-article:16034045 | lifeskim:mentions | umls-concept:C1742737 | lld:lifeskim |
pubmed-article:16034045 | lifeskim:mentions | umls-concept:C0243067 | lld:lifeskim |
pubmed-article:16034045 | lifeskim:mentions | umls-concept:C0678227 | lld:lifeskim |
pubmed-article:16034045 | pubmed:issue | 21 | lld:pubmed |
pubmed-article:16034045 | pubmed:dateCreated | 2005-7-21 | lld:pubmed |
pubmed-article:16034045 | pubmed:abstractText | Hereditary nonpolyposis colorectal cancer (HNPCC) may be caused by mutations in mismatch repair (MMR) genes. The aim of this study was to validate immunohistochemistry and family history as prescreening tools to predict germline mutations in MLH1, MSH2, and MSH6. | lld:pubmed |
pubmed-article:16034045 | pubmed:language | eng | lld:pubmed |
pubmed-article:16034045 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:16034045 | pubmed:citationSubset | IM | lld:pubmed |
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pubmed-article:16034045 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:16034045 | pubmed:month | Jul | lld:pubmed |
pubmed-article:16034045 | pubmed:issn | 0732-183X | lld:pubmed |
pubmed-article:16034045 | pubmed:author | pubmed-author:AaseSteinarS | lld:pubmed |
pubmed-article:16034045 | pubmed:author | pubmed-author:HeimdalKetilK | lld:pubmed |
pubmed-article:16034045 | pubmed:author | pubmed-author:MøllerPålP | lld:pubmed |
pubmed-article:16034045 | pubmed:author | pubmed-author:WijnenJuulJ | lld:pubmed |
pubmed-article:16034045 | pubmed:author | pubmed-author:ApoldJaranJ | lld:pubmed |
pubmed-article:16034045 | pubmed:author | pubmed-author:StormorkenAst... | lld:pubmed |
pubmed-article:16034045 | pubmed:author | pubmed-author:NorènToveT | lld:pubmed |
pubmed-article:16034045 | pubmed:author | pubmed-author:Bowitz-LotheI... | lld:pubmed |
pubmed-article:16034045 | pubmed:author | pubmed-author:KureElinE | lld:pubmed |
pubmed-article:16034045 | pubmed:issnType | Print | lld:pubmed |
pubmed-article:16034045 | pubmed:day | 20 | lld:pubmed |
pubmed-article:16034045 | pubmed:volume | 23 | lld:pubmed |
pubmed-article:16034045 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:16034045 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:16034045 | pubmed:pagination | 4705-12 | lld:pubmed |
pubmed-article:16034045 | pubmed:dateRevised | 2007-11-15 | lld:pubmed |
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pubmed-article:16034045 | pubmed:meshHeading | pubmed-meshheading:16034045... | lld:pubmed |
pubmed-article:16034045 | pubmed:year | 2005 | lld:pubmed |
pubmed-article:16034045 | pubmed:articleTitle | Immunohistochemistry identifies carriers of mismatch repair gene defects causing hereditary nonpolyposis colorectal cancer. | lld:pubmed |
pubmed-article:16034045 | pubmed:affiliation | Section of Genetic Counselling, Department of Cancer Genetics, The Norwegian Radium Hospital, N-0310 Oslo, Norway. | lld:pubmed |
pubmed-article:16034045 | pubmed:publicationType | Journal Article | lld:pubmed |
pubmed-article:16034045 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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