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pubmed-article:15934098pubmed:abstractTextProteolytic autoantigen cleavage by the serine protease granzyme B has been implicated in the development of systemic autoimmune disease; however, there has been no conclusive demonstration of a pathogenic role for granzyme B in autoimmunity. In this study, we evaluated the role of granzyme B in a murine model of autoimmunity.lld:pubmed
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pubmed-article:15934098pubmed:pagination1684-93lld:pubmed
pubmed-article:15934098pubmed:dateRevised2007-11-14lld:pubmed
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pubmed-article:15934098pubmed:articleTitleGranzyme B is dispensable for immunologic tolerance to self in a murine model of systemic lupus erythematosus.lld:pubmed
pubmed-article:15934098pubmed:affiliationStanford University School of Medicine, Stanford, California, USA.lld:pubmed
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pubmed-article:15934098pubmed:publicationTypeResearch Support, U.S. Gov't, P.H.S.lld:pubmed
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