| pubmed-article:15886235 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:15886235 | lifeskim:mentions | umls-concept:C0043210 | lld:lifeskim |
| pubmed-article:15886235 | lifeskim:mentions | umls-concept:C0016658 | lld:lifeskim |
| pubmed-article:15886235 | lifeskim:mentions | umls-concept:C0035647 | lld:lifeskim |
| pubmed-article:15886235 | lifeskim:mentions | umls-concept:C0005938 | lld:lifeskim |
| pubmed-article:15886235 | lifeskim:mentions | umls-concept:C0108082 | lld:lifeskim |
| pubmed-article:15886235 | lifeskim:mentions | umls-concept:C0332281 | lld:lifeskim |
| pubmed-article:15886235 | lifeskim:mentions | umls-concept:C0232970 | lld:lifeskim |
| pubmed-article:15886235 | lifeskim:mentions | umls-concept:C1335671 | lld:lifeskim |
| pubmed-article:15886235 | lifeskim:mentions | umls-concept:C0678951 | lld:lifeskim |
| pubmed-article:15886235 | pubmed:issue | 8 | lld:pubmed |
| pubmed-article:15886235 | pubmed:dateCreated | 2005-8-9 | lld:pubmed |
| pubmed-article:15886235 | pubmed:abstractText | Osteoporosis is a systemic disease with a strong genetic component. Vitamin D receptor (VDR) gene polymorphisms explain only a small part of the genetic influence on the level of bone mineral density (BMD), whereas their effect on fracture remains uncertain. | lld:pubmed |
| pubmed-article:15886235 | pubmed:language | eng | lld:pubmed |
| pubmed-article:15886235 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:15886235 | pubmed:citationSubset | AIM | lld:pubmed |
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| pubmed-article:15886235 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:15886235 | pubmed:month | Aug | lld:pubmed |
| pubmed-article:15886235 | pubmed:issn | 0021-972X | lld:pubmed |
| pubmed-article:15886235 | pubmed:author | pubmed-author:BoreaBB | lld:pubmed |
| pubmed-article:15886235 | pubmed:author | pubmed-author:MunozFF | lld:pubmed |
| pubmed-article:15886235 | pubmed:author | pubmed-author:DelmasP DPD | lld:pubmed |
| pubmed-article:15886235 | pubmed:author | pubmed-author:GarneroPP | lld:pubmed |
| pubmed-article:15886235 | pubmed:author | pubmed-author:Sornay-RenduE... | lld:pubmed |
| pubmed-article:15886235 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:15886235 | pubmed:volume | 90 | lld:pubmed |
| pubmed-article:15886235 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:15886235 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:15886235 | pubmed:pagination | 4829-35 | lld:pubmed |
| pubmed-article:15886235 | pubmed:dateRevised | 2006-11-15 | lld:pubmed |
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| pubmed-article:15886235 | pubmed:meshHeading | pubmed-meshheading:15886235... | lld:pubmed |
| pubmed-article:15886235 | pubmed:year | 2005 | lld:pubmed |
| pubmed-article:15886235 | pubmed:articleTitle | Vitamin D receptor gene polymorphisms are associated with the risk of fractures in postmenopausal women, independently of bone mineral density. | lld:pubmed |
| pubmed-article:15886235 | pubmed:affiliation | Institut National de la Santé et de la Recherche Médicale Research Unit 403, Hôpital E Herriot, Pavillon F, 69437 Lyon Cedex 03, France. patrick.garnero@synarc.com | lld:pubmed |
| pubmed-article:15886235 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:15886235 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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