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| pubmed-article:15843421 | lifeskim:mentions | umls-concept:C0009402 | lld:lifeskim |
| pubmed-article:15843421 | lifeskim:mentions | umls-concept:C0162832 | lld:lifeskim |
| pubmed-article:15843421 | lifeskim:mentions | umls-concept:C0026882 | lld:lifeskim |
| pubmed-article:15843421 | lifeskim:mentions | umls-concept:C0079419 | lld:lifeskim |
| pubmed-article:15843421 | lifeskim:mentions | umls-concept:C0237881 | lld:lifeskim |
| pubmed-article:15843421 | lifeskim:mentions | umls-concept:C0220901 | lld:lifeskim |
| pubmed-article:15843421 | lifeskim:mentions | umls-concept:C0750502 | lld:lifeskim |
| pubmed-article:15843421 | pubmed:issue | 9 | lld:pubmed |
| pubmed-article:15843421 | pubmed:dateCreated | 2005-8-15 | lld:pubmed |
| pubmed-article:15843421 | pubmed:abstractText | Accumulation of molecular alterations, including mutations in Kirsten-ras (K-ras), p53, and adenomatous polyposis coli (APC), contribute to colorectal carcinogenesis. Our group has previously characterised a panel of sporadic colorectal adenocarcinomas for mutations in these three genes and has shown that p53 and K-ras mutations rarely occur in the same colorectal tumour. This suggests that mutations in these genes are on separate pathways to colorectal cancer development and may influence patient prognosis independently. | lld:pubmed |
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| pubmed-article:15843421 | pubmed:language | eng | lld:pubmed |
| pubmed-article:15843421 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:15843421 | pubmed:citationSubset | AIM | lld:pubmed |
| pubmed-article:15843421 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:15843421 | pubmed:month | Sep | lld:pubmed |
| pubmed-article:15843421 | pubmed:issn | 0017-5749 | lld:pubmed |
| pubmed-article:15843421 | pubmed:author | pubmed-author:SmithGG | lld:pubmed |
| pubmed-article:15843421 | pubmed:author | pubmed-author:WolfC RCR | lld:pubmed |
| pubmed-article:15843421 | pubmed:author | pubmed-author:CareyF AFA | lld:pubmed |
| pubmed-article:15843421 | pubmed:author | pubmed-author:SteeleR J CRJ | lld:pubmed |
| pubmed-article:15843421 | pubmed:author | pubmed-author:ConlinAA | lld:pubmed |
| pubmed-article:15843421 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:15843421 | pubmed:volume | 54 | lld:pubmed |
| pubmed-article:15843421 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:15843421 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:15843421 | pubmed:pagination | 1283-6 | lld:pubmed |
| pubmed-article:15843421 | pubmed:dateRevised | 2009-11-18 | lld:pubmed |
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| pubmed-article:15843421 | pubmed:year | 2005 | lld:pubmed |
| pubmed-article:15843421 | pubmed:articleTitle | The prognostic significance of K-ras, p53, and APC mutations in colorectal carcinoma. | lld:pubmed |
| pubmed-article:15843421 | pubmed:affiliation | Biomedical Research Centre, Level 5, Ninewells Hospital and Medical School, Ninewells Ave, Dundee DD1 9SY, UK. abbyconlin75@yahoo.co.uk | lld:pubmed |
| pubmed-article:15843421 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:15843421 | pubmed:publicationType | Comparative Study | lld:pubmed |
| pubmed-article:15843421 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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