| pubmed-article:1580298 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:1580298 | lifeskim:mentions | umls-concept:C0085584 | lld:lifeskim |
| pubmed-article:1580298 | lifeskim:mentions | umls-concept:C0206083 | lld:lifeskim |
| pubmed-article:1580298 | lifeskim:mentions | umls-concept:C1179435 | lld:lifeskim |
| pubmed-article:1580298 | lifeskim:mentions | umls-concept:C1705248 | lld:lifeskim |
| pubmed-article:1580298 | lifeskim:mentions | umls-concept:C1548799 | lld:lifeskim |
| pubmed-article:1580298 | lifeskim:mentions | umls-concept:C1524073 | lld:lifeskim |
| pubmed-article:1580298 | lifeskim:mentions | umls-concept:C0449432 | lld:lifeskim |
| pubmed-article:1580298 | pubmed:issue | 5 | lld:pubmed |
| pubmed-article:1580298 | pubmed:dateCreated | 1992-6-11 | lld:pubmed |
| pubmed-article:1580298 | pubmed:abstractText | Severe hyponatremia is often associated with permanent brain damage. There has been substantial controversy about whether central pontine myelinolysis (CPM), a rare neurologic disorder of uncertain etiology, can complicate either hyponatremia or its therapy. This study was undertaken to determine how often hyponatremic patients with the clinical diagnosis of CPM actually have the disorder as an integral structural component of their encephalopathy. | lld:pubmed |
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| pubmed-article:1580298 | pubmed:language | eng | lld:pubmed |
| pubmed-article:1580298 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:1580298 | pubmed:citationSubset | AIM | lld:pubmed |
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| pubmed-article:1580298 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:1580298 | pubmed:month | May | lld:pubmed |
| pubmed-article:1580298 | pubmed:issn | 0002-9343 | lld:pubmed |
| pubmed-article:1580298 | pubmed:author | pubmed-author:ArieffA IAI | lld:pubmed |
| pubmed-article:1580298 | pubmed:author | pubmed-author:WasikAA | lld:pubmed |
| pubmed-article:1580298 | pubmed:author | pubmed-author:KucharczykJJ | lld:pubmed |
| pubmed-article:1580298 | pubmed:author | pubmed-author:KucharczykWW | lld:pubmed |
| pubmed-article:1580298 | pubmed:author | pubmed-author:TienRR | lld:pubmed |
| pubmed-article:1580298 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:1580298 | pubmed:volume | 92 | lld:pubmed |
| pubmed-article:1580298 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:1580298 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:1580298 | pubmed:pagination | 513-22 | lld:pubmed |
| pubmed-article:1580298 | pubmed:dateRevised | 2007-11-14 | lld:pubmed |
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| pubmed-article:1580298 | pubmed:meshHeading | pubmed-meshheading:1580298-... | lld:pubmed |
| pubmed-article:1580298 | pubmed:meshHeading | pubmed-meshheading:1580298-... | lld:pubmed |
| pubmed-article:1580298 | pubmed:year | 1992 | lld:pubmed |
| pubmed-article:1580298 | pubmed:articleTitle | Hyponatremic encephalopathy: is central pontine myelinolysis a component? | lld:pubmed |
| pubmed-article:1580298 | pubmed:affiliation | Neuroradiology Section, University of California, San Francisco. | lld:pubmed |
| pubmed-article:1580298 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:1580298 | pubmed:publicationType | Research Support, U.S. Gov't, P.H.S. | lld:pubmed |
| pubmed-article:1580298 | pubmed:publicationType | Research Support, U.S. Gov't, Non-P.H.S. | lld:pubmed |
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