| pubmed-article:15765131 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:15765131 | lifeskim:mentions | umls-concept:C0018801 | lld:lifeskim |
| pubmed-article:15765131 | lifeskim:mentions | umls-concept:C0242606 | lld:lifeskim |
| pubmed-article:15765131 | lifeskim:mentions | umls-concept:C0030054 | lld:lifeskim |
| pubmed-article:15765131 | lifeskim:mentions | umls-concept:C0242184 | lld:lifeskim |
| pubmed-article:15765131 | pubmed:issue | 3 | lld:pubmed |
| pubmed-article:15765131 | pubmed:dateCreated | 2005-3-14 | lld:pubmed |
| pubmed-article:15765131 | pubmed:abstractText | A constant supply of oxygen is indispensable for cardiac viability and function. However, the role of oxygen and oxygen-associated processes in the heart is complex, and they and can be either beneficial or contribute to cardiac dysfunction and death. As oxygen is a major determinant of cardiac gene expression, and a critical participant in the formation of ROS and numerous other cellular processes, consideration of its role in the heart is essential in understanding the pathogenesis of cardiac dysfunction. | lld:pubmed |
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