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pubmed-article:15679584pubmed:abstractTextThe pathogenic mechanisms of UV-induced skin lesions of lupus erythematosus (LE) are unknown. In a recent study of pathogenic mechanisms of polymorphic light eruption (PLE), significantly more Langerhans cells (LCs) persisted in the epidermis after UVB overexposure than in healthy individuals. Interestingly, the same phenomenon was observed in one subacute cutaneous lupus erythematosus (SCLE) patient. It could therefore be hypothesized that both photodermatoses share a common pathogenic mechanism of photosensitivity. In the present study, we tested this hypothesis by investigating leucocyte trafficking in the initial phase of cutaneous LE after intense UVB exposure.lld:pubmed
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pubmed-article:15679584pubmed:dateRevised2006-11-15lld:pubmed
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pubmed-article:15679584pubmed:articleTitleUVB-induced leucocyte trafficking in the epidermis of photosensitive lupus erythematosus patients: normal depletion of Langerhans cells.lld:pubmed
pubmed-article:15679584pubmed:affiliationDepartment of Dermatology, Leiden University Medical Center, Leiden, The Netherlands. a.s.janssens@lumc.nllld:pubmed
pubmed-article:15679584pubmed:publicationTypeJournal Articlelld:pubmed
pubmed-article:15679584pubmed:publicationTypeResearch Support, Non-U.S. Gov'tlld:pubmed
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