pubmed-article:15667561 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:15667561 | lifeskim:mentions | umls-concept:C0597357 | lld:lifeskim |
pubmed-article:15667561 | lifeskim:mentions | umls-concept:C0385569 | lld:lifeskim |
pubmed-article:15667561 | pubmed:issue | 2 | lld:pubmed |
pubmed-article:15667561 | pubmed:dateCreated | 2005-1-25 | lld:pubmed |
pubmed-article:15667561 | pubmed:abstractText | Interleukin 24 (IL-24) is a new member of the IL-10 family of cytokines and it signals through two heterodimeric receptors: IL-20R1/IL-20R2 and IL-22R1/IL-20R2. Upon binding to its receptors, IL-24 induces rapid activation of Stat-1 and Stat-3 transcription factors, which appear to play a role in cell survival and proliferation. Under physiological conditions, the major sources of IL-24 are the activated monocytes and T helper 2 cells, whereas the major IL-24 target tissues, based on the receptor expression pattern, are non-haematopoietic in origin, and include skin, lung and reproductive tissues. Structurally and functionally, IL-24 is highly conserved across species. This review highlights our current knowledge of IL-24 as a cytokine, with much less emphasis placed on the non-receptor-mediated functions (a subject of several reviews) focused on in much of the earlier literature on IL-24. The potential roles of IL-24 as part of a complex cytokine network in wound healing, psoriasis and cancer are discussed. | lld:pubmed |
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pubmed-article:15667561 | pubmed:language | eng | lld:pubmed |
pubmed-article:15667561 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:15667561 | pubmed:citationSubset | IM | lld:pubmed |
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pubmed-article:15667561 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:15667561 | pubmed:month | Feb | lld:pubmed |
pubmed-article:15667561 | pubmed:issn | 0019-2805 | lld:pubmed |
pubmed-article:15667561 | pubmed:author | pubmed-author:WeilT STS | lld:pubmed |
pubmed-article:15667561 | pubmed:author | pubmed-author:LiangPengP | lld:pubmed |
pubmed-article:15667561 | pubmed:issnType | Print | lld:pubmed |
pubmed-article:15667561 | pubmed:volume | 114 | lld:pubmed |
pubmed-article:15667561 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:15667561 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:15667561 | pubmed:pagination | 166-70 | lld:pubmed |
pubmed-article:15667561 | pubmed:dateRevised | 2009-11-18 | lld:pubmed |
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pubmed-article:15667561 | pubmed:year | 2005 | lld:pubmed |
pubmed-article:15667561 | pubmed:articleTitle | Interleukin-24 and its receptors. | lld:pubmed |
pubmed-article:15667561 | pubmed:affiliation | Department of Cancer Biology, Vanderbilt University School of Medicine, Nashville, TN 37232, USA. | lld:pubmed |
pubmed-article:15667561 | pubmed:publicationType | Journal Article | lld:pubmed |
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