pubmed-article:15599934 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:15599934 | lifeskim:mentions | umls-concept:C0332307 | lld:lifeskim |
pubmed-article:15599934 | lifeskim:mentions | umls-concept:C0349536 | lld:lifeskim |
pubmed-article:15599934 | lifeskim:mentions | umls-concept:C0597357 | lld:lifeskim |
pubmed-article:15599934 | lifeskim:mentions | umls-concept:C0040690 | lld:lifeskim |
pubmed-article:15599934 | lifeskim:mentions | umls-concept:C0332197 | lld:lifeskim |
pubmed-article:15599934 | lifeskim:mentions | umls-concept:C0920269 | lld:lifeskim |
pubmed-article:15599934 | lifeskim:mentions | umls-concept:C0596611 | lld:lifeskim |
pubmed-article:15599934 | pubmed:issue | 2 | lld:pubmed |
pubmed-article:15599934 | pubmed:dateCreated | 2005-1-17 | lld:pubmed |
pubmed-article:15599934 | pubmed:abstractText | Microsatellite instability (MSI) with concomitant mutations in the coding region of transforming growth factor-beta type II receptor (TGFbetaRII) results in an aberrant growth-regulatory phenotype in colorectal carcinomas. The authors postulated that a similar mechanism occurred during the malignant evolution of small bowel carcinoid tumors. | lld:pubmed |
pubmed-article:15599934 | pubmed:language | eng | lld:pubmed |
pubmed-article:15599934 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:15599934 | pubmed:citationSubset | AIM | lld:pubmed |
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pubmed-article:15599934 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:15599934 | pubmed:month | Jan | lld:pubmed |
pubmed-article:15599934 | pubmed:issn | 0008-543X | lld:pubmed |
pubmed-article:15599934 | pubmed:author | pubmed-author:CampRobert... | lld:pubmed |
pubmed-article:15599934 | pubmed:author | pubmed-author:ManeShrikant... | lld:pubmed |
pubmed-article:15599934 | pubmed:author | pubmed-author:KiddMarkM | lld:pubmed |
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pubmed-article:15599934 | pubmed:author | pubmed-author:ShapiroMichae... | lld:pubmed |
pubmed-article:15599934 | pubmed:author | pubmed-author:EickGeetaG | lld:pubmed |
pubmed-article:15599934 | pubmed:copyrightInfo | (c) 2004 American Cancer Society. | lld:pubmed |
pubmed-article:15599934 | pubmed:issnType | Print | lld:pubmed |
pubmed-article:15599934 | pubmed:day | 15 | lld:pubmed |
pubmed-article:15599934 | pubmed:volume | 103 | lld:pubmed |
pubmed-article:15599934 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:15599934 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:15599934 | pubmed:pagination | 229-36 | lld:pubmed |
pubmed-article:15599934 | pubmed:dateRevised | 2007-11-15 | lld:pubmed |
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pubmed-article:15599934 | pubmed:year | 2005 | lld:pubmed |
pubmed-article:15599934 | pubmed:articleTitle | Microsatellite instability and gene mutations in transforming growth factor-beta type II receptor are absent in small bowel carcinoid tumors. | lld:pubmed |
pubmed-article:15599934 | pubmed:affiliation | Department of Surgery, Yale University School of Medicine, New Haven, Connecticut 06520-8062, USA. mkidd01@snet.net | lld:pubmed |
pubmed-article:15599934 | pubmed:publicationType | Journal Article | lld:pubmed |
pubmed-article:15599934 | pubmed:publicationType | Comparative Study | lld:pubmed |
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