pubmed-article:15569977 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:15569977 | lifeskim:mentions | umls-concept:C0033325 | lld:lifeskim |
pubmed-article:15569977 | lifeskim:mentions | umls-concept:C0032854 | lld:lifeskim |
pubmed-article:15569977 | lifeskim:mentions | umls-concept:C0009402 | lld:lifeskim |
pubmed-article:15569977 | lifeskim:mentions | umls-concept:C0007589 | lld:lifeskim |
pubmed-article:15569977 | lifeskim:mentions | umls-concept:C1655731 | lld:lifeskim |
pubmed-article:15569977 | lifeskim:mentions | umls-concept:C1415857 | lld:lifeskim |
pubmed-article:15569977 | lifeskim:mentions | umls-concept:C1561558 | lld:lifeskim |
pubmed-article:15569977 | lifeskim:mentions | umls-concept:C1511938 | lld:lifeskim |
pubmed-article:15569977 | pubmed:issue | 22 | lld:pubmed |
pubmed-article:15569977 | pubmed:dateCreated | 2004-11-30 | lld:pubmed |
pubmed-article:15569977 | pubmed:abstractText | ID4 gene is a member of the inhibitor of DNA binding (ID) family proteins that inhibit DNA binding of basic helix-loop-helix transcription factors. The epigenetic inactivation of ID4 gene on colorectal cancer (CRC) development and its clinical significance was assessed. | lld:pubmed |
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pubmed-article:15569977 | pubmed:language | eng | lld:pubmed |
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pubmed-article:15569977 | pubmed:citationSubset | IM | lld:pubmed |
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pubmed-article:15569977 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:15569977 | pubmed:month | Nov | lld:pubmed |
pubmed-article:15569977 | pubmed:issn | 1078-0432 | lld:pubmed |
pubmed-article:15569977 | pubmed:author | pubmed-author:HoonDave S... | lld:pubmed |
pubmed-article:15569977 | pubmed:author | pubmed-author:BilchikAnton... | lld:pubmed |
pubmed-article:15569977 | pubmed:author | pubmed-author:ShinozakiMasa... | lld:pubmed |
pubmed-article:15569977 | pubmed:author | pubmed-author:FujimotoAkihi... | lld:pubmed |
pubmed-article:15569977 | pubmed:author | pubmed-author:TakeuchiHiroy... | lld:pubmed |
pubmed-article:15569977 | pubmed:author | pubmed-author:UmetaniNaoyuk... | lld:pubmed |
pubmed-article:15569977 | pubmed:issnType | Print | lld:pubmed |
pubmed-article:15569977 | pubmed:day | 15 | lld:pubmed |
pubmed-article:15569977 | pubmed:volume | 10 | lld:pubmed |
pubmed-article:15569977 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:15569977 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:15569977 | pubmed:pagination | 7475-83 | lld:pubmed |
pubmed-article:15569977 | pubmed:dateRevised | 2007-11-14 | lld:pubmed |
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pubmed-article:15569977 | pubmed:year | 2004 | lld:pubmed |
pubmed-article:15569977 | pubmed:articleTitle | Epigenetic inactivation of ID4 in colorectal carcinomas correlates with poor differentiation and unfavorable prognosis. | lld:pubmed |
pubmed-article:15569977 | pubmed:affiliation | Department of Molecular Oncology, and Division of Gastrointestinal Oncology, John Wayne Cancer Institute, Santa Monica, California 90404, USA. | lld:pubmed |
pubmed-article:15569977 | pubmed:publicationType | Journal Article | lld:pubmed |
pubmed-article:15569977 | pubmed:publicationType | Research Support, U.S. Gov't, P.H.S. | lld:pubmed |
pubmed-article:15569977 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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