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pubmed-article:1556774pubmed:abstractTextEstrogens are potent mammary tumor promoters influencing post-initiation events via epigenetic mechanisms. The upregulation (i.e., induction) of the C16 alpha-hydroxylation pathway during 17 beta-estradiol (E2) biotransformation has been associated with mammary cell transformation. The action of E2 metabolites on tumorigenic transformation, however, is poorly understood.lld:pubmed
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pubmed-article:1556774pubmed:articleTitleInduction by estrogen metabolite 16 alpha-hydroxyestrone of genotoxic damage and aberrant proliferation in mouse mammary epithelial cells.lld:pubmed
pubmed-article:1556774pubmed:affiliationBreast Cancer Research Laboratory, Memorial Sloan-Kettering Cancer Center, New York, NY 10021.lld:pubmed
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