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pubmed-article:15557916pubmed:abstractTextInflammation is involved in the pathogenesis of atherosclerosis and abdominal aortic aneurysm (AAA), and chemokines are mediators of the inflammatory process. The homozygous Delta 32 deletion mutation of the gene of the chemokine receptor CCR5 is a cause of its lack in inflammatory cells. The purpose of this study was to investigate the relationship between CCR5 Delta 32 deletion mutation and AAA, peripheral arterial occlusive disease (PAOD), and carotid stenosis.lld:pubmed
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pubmed-article:15557916pubmed:articleTitleGenetic risk factor characterizes abdominal aortic aneurysm from arterial occlusive disease in human beings: CCR5 Delta 32 deletion.lld:pubmed
pubmed-article:15557916pubmed:affiliationDipartimento MCO, Clinica Chirurgica Generale, Università degli Studi di Milano-Polo Didattico S. Paolo, 9 I-20142 Milano, Italy. giorgio.ghilardi@unimi.itlld:pubmed
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