| pubmed-article:1555637 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:1555637 | lifeskim:mentions | umls-concept:C1882561 | lld:lifeskim |
| pubmed-article:1555637 | lifeskim:mentions | umls-concept:C0042401 | lld:lifeskim |
| pubmed-article:1555637 | lifeskim:mentions | umls-concept:C0006669 | lld:lifeskim |
| pubmed-article:1555637 | lifeskim:mentions | umls-concept:C1157570 | lld:lifeskim |
| pubmed-article:1555637 | lifeskim:mentions | umls-concept:C0599946 | lld:lifeskim |
| pubmed-article:1555637 | lifeskim:mentions | umls-concept:C0243077 | lld:lifeskim |
| pubmed-article:1555637 | pubmed:issue | 1 | lld:pubmed |
| pubmed-article:1555637 | pubmed:dateCreated | 1992-5-7 | lld:pubmed |
| pubmed-article:1555637 | pubmed:abstractText | The effect of inhibitors of nitric oxide synthesis were examined on the endothelium-dependent relaxation induced by acetylcholine and human alpha-calcitonin gene-related peptide (CGRP) on rat isolated aortic rings preconstricted with noradrenaline. The endothelium-dependent vasorelaxation induced by acetylcholine and CGRP was inhibited by NG-monomethyl-L-arginine (L-NMMA) and NG-nitro-L-arginine (L-NOARG) in a concentration-related fashion. The inhibition of endothelium-dependent relaxation by L-NMMA and L-NOARG was partially reversed by L-arginine but not by D-arginine for both acetylcholine and CGRP. The data presented suggests that CGRP, like acetylcholine; relaxes the rat aorta via the release of nitric oxide. | lld:pubmed |
| pubmed-article:1555637 | pubmed:language | eng | lld:pubmed |
| pubmed-article:1555637 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:1555637 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:1555637 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:1555637 | pubmed:month | Feb | lld:pubmed |
| pubmed-article:1555637 | pubmed:issn | 0014-2999 | lld:pubmed |
| pubmed-article:1555637 | pubmed:author | pubmed-author:MarshallII | lld:pubmed |
| pubmed-article:1555637 | pubmed:author | pubmed-author:GrayD WDW | lld:pubmed |
| pubmed-article:1555637 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:1555637 | pubmed:day | 25 | lld:pubmed |
| pubmed-article:1555637 | pubmed:volume | 212 | lld:pubmed |
| pubmed-article:1555637 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:1555637 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:1555637 | pubmed:pagination | 37-42 | lld:pubmed |
| pubmed-article:1555637 | pubmed:dateRevised | 2006-11-15 | lld:pubmed |
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| pubmed-article:1555637 | pubmed:year | 1992 | lld:pubmed |
| pubmed-article:1555637 | pubmed:articleTitle | Nitric oxide synthesis inhibitors attenuate calcitonin gene-related peptide endothelium-dependent vasorelaxation in rat aorta. | lld:pubmed |
| pubmed-article:1555637 | pubmed:affiliation | Department of Pharmacology, University College and Middlesex School of Medicine, University College London, U.K. | lld:pubmed |
| pubmed-article:1555637 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:1555637 | pubmed:publicationType | In Vitro | lld:pubmed |
| pubmed-article:1555637 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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