| pubmed-article:15331400 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:15331400 | lifeskim:mentions | umls-concept:C0021368 | lld:lifeskim |
| pubmed-article:15331400 | lifeskim:mentions | umls-concept:C0043240 | lld:lifeskim |
| pubmed-article:15331400 | lifeskim:mentions | umls-concept:C0521457 | lld:lifeskim |
| pubmed-article:15331400 | lifeskim:mentions | umls-concept:C0387583 | lld:lifeskim |
| pubmed-article:15331400 | lifeskim:mentions | umls-concept:C0127400 | lld:lifeskim |
| pubmed-article:15331400 | pubmed:issue | 3 | lld:pubmed |
| pubmed-article:15331400 | pubmed:dateCreated | 2004-8-30 | lld:pubmed |
| pubmed-article:15331400 | pubmed:abstractText | Cyclooxygenase-2 (COX-2) and the prostaglandin products generated as a result of COX-2 activity mediate a variety of biological and pathological processes. Scarless healing occurs in fetal skin in the first and second trimesters of development. This scarless healing process is known to proceed without a significant inflammatory response, which appears to be important for the lack of scarring. Because the COX-2 pathway is an integral component of inflammation, we investigated its role in the fetal repair process using a mouse model of scarless fetal wound healing. COX-2 expression in scarless and fibrotic fetal wounds was examined. In addition, the ability of exogenous prostaglandin E(2) to alter scarless fetal healing was evaluated. The results suggest that the COX-2 pathway is involved in scar production in fetal skin and that targeting COX-2 may be useful for limiting scar formation in adult skin. | lld:pubmed |
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| pubmed-article:15331400 | pubmed:language | eng | lld:pubmed |
| pubmed-article:15331400 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:15331400 | pubmed:citationSubset | AIM | lld:pubmed |
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| pubmed-article:15331400 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:15331400 | pubmed:month | Sep | lld:pubmed |
| pubmed-article:15331400 | pubmed:issn | 0002-9440 | lld:pubmed |
| pubmed-article:15331400 | pubmed:author | pubmed-author:FlavahanNicho... | lld:pubmed |
| pubmed-article:15331400 | pubmed:author | pubmed-author:WilgusTraci... | lld:pubmed |
| pubmed-article:15331400 | pubmed:author | pubmed-author:OberyszynTati... | lld:pubmed |
| pubmed-article:15331400 | pubmed:author | pubmed-author:MitraSrabaniS | lld:pubmed |
| pubmed-article:15331400 | pubmed:author | pubmed-author:HillKara JKJ | lld:pubmed |
| pubmed-article:15331400 | pubmed:author | pubmed-author:ToberKathleen... | lld:pubmed |
| pubmed-article:15331400 | pubmed:author | pubmed-author:BergdallValer... | lld:pubmed |
| pubmed-article:15331400 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:15331400 | pubmed:volume | 165 | lld:pubmed |
| pubmed-article:15331400 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:15331400 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:15331400 | pubmed:pagination | 753-61 | lld:pubmed |
| pubmed-article:15331400 | pubmed:dateRevised | 2010-9-21 | lld:pubmed |
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