pubmed-article:15326261 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:15326261 | lifeskim:mentions | umls-concept:C0004936 | lld:lifeskim |
pubmed-article:15326261 | lifeskim:mentions | umls-concept:C0497327 | lld:lifeskim |
pubmed-article:15326261 | lifeskim:mentions | umls-concept:C0876926 | lld:lifeskim |
pubmed-article:15326261 | lifeskim:mentions | umls-concept:C0681842 | lld:lifeskim |
pubmed-article:15326261 | pubmed:issue | 4 | lld:pubmed |
pubmed-article:15326261 | pubmed:dateCreated | 2004-8-24 | lld:pubmed |
pubmed-article:15326261 | pubmed:abstractText | The authors studied the association between APOE-epsilon4 genotype and axis I and II psychiatric disorders an average of 30 years after traumatic brain injury. Sixty patients were dichotomized into subjects with and without APOE-epsilon4 allele. Dementia and subclinical dementia were significantly more common with the presence of APOE-epsilon4. The occurrence of other psychiatric disorders did not differ between patients with and without APOE-epsilon4 allele. | lld:pubmed |
pubmed-article:15326261 | pubmed:language | eng | lld:pubmed |
pubmed-article:15326261 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:15326261 | pubmed:citationSubset | AIM | lld:pubmed |
pubmed-article:15326261 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:15326261 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:15326261 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:15326261 | pubmed:month | Aug | lld:pubmed |
pubmed-article:15326261 | pubmed:issn | 1526-632X | lld:pubmed |
pubmed-article:15326261 | pubmed:author | pubmed-author:TenovuoOO | lld:pubmed |
pubmed-article:15326261 | pubmed:author | pubmed-author:IsoniemiHH | lld:pubmed |
pubmed-article:15326261 | pubmed:author | pubmed-author:TaiminenTT | lld:pubmed |
pubmed-article:15326261 | pubmed:author | pubmed-author:HinkkaSS | lld:pubmed |
pubmed-article:15326261 | pubmed:author | pubmed-author:PortinRR | lld:pubmed |
pubmed-article:15326261 | pubmed:author | pubmed-author:KairistoVV | lld:pubmed |
pubmed-article:15326261 | pubmed:author | pubmed-author:KoponenSS | lld:pubmed |
pubmed-article:15326261 | pubmed:issnType | Electronic | lld:pubmed |
pubmed-article:15326261 | pubmed:day | 24 | lld:pubmed |
pubmed-article:15326261 | pubmed:volume | 63 | lld:pubmed |
pubmed-article:15326261 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:15326261 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:15326261 | pubmed:pagination | 749-50 | lld:pubmed |
pubmed-article:15326261 | pubmed:dateRevised | 2006-11-15 | lld:pubmed |
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pubmed-article:15326261 | pubmed:meshHeading | pubmed-meshheading:15326261... | lld:pubmed |
pubmed-article:15326261 | pubmed:year | 2004 | lld:pubmed |
pubmed-article:15326261 | pubmed:articleTitle | APOE-epsilon4 predicts dementia but not other psychiatric disorders after traumatic brain injury. | lld:pubmed |
pubmed-article:15326261 | pubmed:affiliation | Department of Psychiatry, Turku University Hospital, PL 52, FIN-20521 Turku, Finland. salla.koponen@utu.fi | lld:pubmed |
pubmed-article:15326261 | pubmed:publicationType | Journal Article | lld:pubmed |
pubmed-article:15326261 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
entrez-gene:348 | entrezgene:pubmed | pubmed-article:15326261 | lld:entrezgene |
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