pubmed-article:15326234 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:15326234 | lifeskim:mentions | umls-concept:C0035647 | lld:lifeskim |
pubmed-article:15326234 | lifeskim:mentions | umls-concept:C0013421 | lld:lifeskim |
pubmed-article:15326234 | lifeskim:mentions | umls-concept:C0026882 | lld:lifeskim |
pubmed-article:15326234 | lifeskim:mentions | umls-concept:C1851945 | lld:lifeskim |
pubmed-article:15326234 | lifeskim:mentions | umls-concept:C0154409 | lld:lifeskim |
pubmed-article:15326234 | lifeskim:mentions | umls-concept:C1540039 | lld:lifeskim |
pubmed-article:15326234 | lifeskim:mentions | umls-concept:C0205217 | lld:lifeskim |
pubmed-article:15326234 | lifeskim:mentions | umls-concept:C1742737 | lld:lifeskim |
pubmed-article:15326234 | pubmed:issue | 4 | lld:pubmed |
pubmed-article:15326234 | pubmed:dateCreated | 2004-8-24 | lld:pubmed |
pubmed-article:15326234 | pubmed:abstractText | Prior studies suggest that dystonia is comorbid with affective disorders. This comorbidity could be a reaction to a chronic debilitating disorder or expression of a predisposing gene. The authors took advantage of the identification of a gene for dystonia, DYT1, to test these alternative explanations. | lld:pubmed |
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pubmed-article:15326234 | pubmed:language | eng | lld:pubmed |
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pubmed-article:15326234 | pubmed:citationSubset | AIM | lld:pubmed |
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pubmed-article:15326234 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:15326234 | pubmed:month | Aug | lld:pubmed |
pubmed-article:15326234 | pubmed:issn | 1526-632X | lld:pubmed |
pubmed-article:15326234 | pubmed:author | pubmed-author:BressmanS BSB | lld:pubmed |
pubmed-article:15326234 | pubmed:author | pubmed-author:OzeliusL JLJ | lld:pubmed |
pubmed-article:15326234 | pubmed:author | pubmed-author:HeimanG AGA | lld:pubmed |
pubmed-article:15326234 | pubmed:author | pubmed-author:OttmanRR | lld:pubmed |
pubmed-article:15326234 | pubmed:author | pubmed-author:RischN JNJ | lld:pubmed |
pubmed-article:15326234 | pubmed:author | pubmed-author:Saunders-Pull... | lld:pubmed |
pubmed-article:15326234 | pubmed:issnType | Electronic | lld:pubmed |
pubmed-article:15326234 | pubmed:day | 24 | lld:pubmed |
pubmed-article:15326234 | pubmed:volume | 63 | lld:pubmed |
pubmed-article:15326234 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:15326234 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:15326234 | pubmed:pagination | 631-7 | lld:pubmed |
pubmed-article:15326234 | pubmed:dateRevised | 2007-11-14 | lld:pubmed |
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pubmed-article:15326234 | pubmed:year | 2004 | lld:pubmed |
pubmed-article:15326234 | pubmed:articleTitle | Increased risk for recurrent major depression in DYT1 dystonia mutation carriers. | lld:pubmed |
pubmed-article:15326234 | pubmed:affiliation | Department of Epidemiology of Joseph L. Mailman School of Public Health, Columbia University, New York 10032, USA. gah13@columbia.edu | lld:pubmed |
pubmed-article:15326234 | pubmed:publicationType | Journal Article | lld:pubmed |
pubmed-article:15326234 | pubmed:publicationType | Comparative Study | lld:pubmed |
pubmed-article:15326234 | pubmed:publicationType | Research Support, U.S. Gov't, P.H.S. | lld:pubmed |
pubmed-article:15326234 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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