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| pubmed-article:15296513 | lifeskim:mentions | umls-concept:C0017337 | lld:lifeskim |
| pubmed-article:15296513 | lifeskim:mentions | umls-concept:C0295921 | lld:lifeskim |
| pubmed-article:15296513 | lifeskim:mentions | umls-concept:C1420120 | lld:lifeskim |
| pubmed-article:15296513 | lifeskim:mentions | umls-concept:C0004083 | lld:lifeskim |
| pubmed-article:15296513 | lifeskim:mentions | umls-concept:C1882417 | lld:lifeskim |
| pubmed-article:15296513 | lifeskim:mentions | umls-concept:C2603343 | lld:lifeskim |
| pubmed-article:15296513 | pubmed:dateCreated | 2004-8-26 | lld:pubmed |
| pubmed-article:15296513 | pubmed:abstractText | The glutamatergic dysfunction hypothesis of schizophrenia suggests that genes involved in glutametergic transmission are candidates for schizophrenic susceptibility genes. We have been performing systematic association studies of schizophrenia with the glutamate receptor and transporter genes. In this study we report an association study of the excitatory amino acid transporter 2 gene, SLC1A2 with schizophrenia. | lld:pubmed |
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| pubmed-article:15296513 | pubmed:language | eng | lld:pubmed |
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| pubmed-article:15296513 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:15296513 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:15296513 | pubmed:issn | 1471-244X | lld:pubmed |
| pubmed-article:15296513 | pubmed:author | pubmed-author:TashiroNobuta... | lld:pubmed |
| pubmed-article:15296513 | pubmed:author | pubmed-author:OzakiNorioN | lld:pubmed |
| pubmed-article:15296513 | pubmed:author | pubmed-author:FukumakiYasuy... | lld:pubmed |
| pubmed-article:15296513 | pubmed:author | pubmed-author:ShibataHiroki... | lld:pubmed |
| pubmed-article:15296513 | pubmed:author | pubmed-author:NinomiyaHidea... | lld:pubmed |
| pubmed-article:15296513 | pubmed:author | pubmed-author:IwataNakaoN | lld:pubmed |
| pubmed-article:15296513 | pubmed:author | pubmed-author:DengXiangdong... | lld:pubmed |
| pubmed-article:15296513 | pubmed:issnType | Electronic | lld:pubmed |
| pubmed-article:15296513 | pubmed:volume | 4 | lld:pubmed |
| pubmed-article:15296513 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:15296513 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:15296513 | pubmed:pagination | 21 | lld:pubmed |
| pubmed-article:15296513 | pubmed:dateRevised | 2009-11-18 | lld:pubmed |
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| pubmed-article:15296513 | pubmed:year | 2004 | lld:pubmed |
| pubmed-article:15296513 | pubmed:articleTitle | Association study of polymorphisms in the excitatory amino acid transporter 2 gene (SLC1A2) with schizophrenia. | lld:pubmed |
| pubmed-article:15296513 | pubmed:affiliation | Division of Disease Genes, Research Center for Genetic Information, Medical Institute of Bioregulation, Kyushu University, Fukuoka 812-8582, Japan. luckbird@gen.kyushu-u.ac.jp | lld:pubmed |
| pubmed-article:15296513 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:15296513 | pubmed:publicationType | Comparative Study | lld:pubmed |
| pubmed-article:15296513 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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